Hybrid computational modeling demonstrates the utility of simulating complex cellular networks in type 1 diabetes.
Persistent destruction of pancreatic β-cells in type 1 diabetes (T1D) results from multifaceted pancreatic cellular interactions in various phase progressions. Owing to the inherent heterogeneity of coupled nonlinear systems, computational modeling based on T1D etiology help achieve a systematic und...
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Auteurs principaux: | , , |
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Format: | article |
Langue: | EN |
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Public Library of Science (PLoS)
2021
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Accès en ligne: | https://doaj.org/article/04fcaf35a1c242a8ba03b34f748257c7 |
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