Compensatory Vasodilator Mechanisms in the Ophthalmic Artery of Endothelial Nitric Oxide Synthase Gene Knockout Mice
Abstract Nitric oxide (NO) generated by endothelial nitric oxide synthase (eNOS) plays an important role in the maintenance of ocular vascular homeostasis. Therefore, perturbations in vascular NO synthesis have been implicated in the pathogenesis of several ocular diseases. We recently reported that...
Guardado en:
Autores principales: | Caroline Manicam, Natalja Ginter, Huige Li, Ning Xia, Evgeny Goloborodko, Jenia Kouchek Zadeh, Aytan Musayeva, Norbert Pfeiffer, Adrian Gericke |
---|---|
Formato: | article |
Lenguaje: | EN |
Publicado: |
Nature Portfolio
2017
|
Materias: | |
Acceso en línea: | https://doaj.org/article/06285f9a5ab14f8f926e07fd906afa12 |
Etiquetas: |
Agregar Etiqueta
Sin Etiquetas, Sea el primero en etiquetar este registro!
|
Ejemplares similares
-
The M1 muscarinic acetylcholine receptor subtype is important for retinal neuron survival in aging mice
por: Panagiotis Laspas, et al.
Publicado: (2019) -
High resistance to Toxoplasma gondii infection in inducible nitric oxide synthase knockout rats
por: Zhen-Jie Wang, et al.
Publicado: (2021) -
Compensatory role of endogenous sulfur dioxide in nitric oxide deficiency-induced hypertension
por: Yunjia Song, et al.
Publicado: (2021) -
Compensatory feto-placental upregulation of the nitric oxide system during fetal growth restriction.
por: Silvia Pisaneschi, et al.
Publicado: (2012) -
Mechanism of inducible nitric oxide synthase exclusion from mycobacterial phagosomes.
por: Alexander S Davis, et al.
Publicado: (2007)