Protein-losing enteropathy managed with percutaneous enlargement of a restrictive atrial septal defect

Protein-losing enteropathy is one of the most feared complications of the Fontan circulation. The diagnosis of protein-losing enteropathy in this setting should prompt a thorough investigation for the presence of a treatable hemodynamic impairment. In this report, we describe a complete reversal of...

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Autores principales:	Milan Djukic, Stefan A. Djordjevic, Andrija S. Pavlovic, Igor Stefanovic, Mirko Topalovic, Ingo Dähnert
Formato:	article
Lenguaje:	EN
Publicado:	Elsevier 2021
Materias:	Procedimento de Fontan Enteropatia por perda de proteínas Septo auricular Cateterismo cardíaco Estenose mitral Dupla saída do ventrículo direito Diseases of the circulatory (Cardiovascular) system RC666-701
Acceso en línea:	https://doaj.org/article/088b12f4e1774c03995d770abcc830dd
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spelling	oai:doaj.org-article:088b12f4e1774c03995d770abcc830dd2021-12-02T05:00:32ZProtein-losing enteropathy managed with percutaneous enlargement of a restrictive atrial septal defect2174-204910.1016/j.repce.2021.11.005https://doaj.org/article/088b12f4e1774c03995d770abcc830dd2021-11-01T00:00:00Zhttp://www.sciencedirect.com/science/article/pii/S2174204921003573https://doaj.org/toc/2174-2049Protein-losing enteropathy is one of the most feared complications of the Fontan circulation. The diagnosis of protein-losing enteropathy in this setting should prompt a thorough investigation for the presence of a treatable hemodynamic impairment. In this report, we describe a complete reversal of protein-losing enteropathy following percutaneous enlargement of a restrictive atrial septal defect in a patient with a fenestrated lateral tunnel Fontan and severe mitral stenosis. Resumo: A enteropatia por perda de proteínas é uma das complicações mais temidas da circulação de Fontan. O diagnóstico de enteropatia por perda de proteínas neste contexto deve suscitar uma avaliação exaustiva da eventual presença de uma alteraçao hemodinâmica tratável. Neste artigo, apresentamos uma completa regressão de enteropatia por perda de proteínas após dilatação percutânea de defeito restritivo do septo interauricular num doente com tunel lateral fenestrado de Fontan e estenose mitral grave.Milan DjukicStefan A. DjordjevicAndrija S. PavlovicIgor StefanovicMirko TopalovicIngo DähnertElsevierarticleProcedimento de FontanEnteropatia por perda de proteínasSepto auricularCateterismo cardíacoEstenose mitralDupla saída do ventrículo direitoDiseases of the circulatory (Cardiovascular) systemRC666-701ENRevista Portuguesa de Cardiologia (English Edition), Vol 40, Iss 11, Pp 895.e1-895.e4 (2021)
institution	DOAJ
collection	DOAJ
language	EN
topic	Procedimento de Fontan Enteropatia por perda de proteínas Septo auricular Cateterismo cardíaco Estenose mitral Dupla saída do ventrículo direito Diseases of the circulatory (Cardiovascular) system RC666-701
spellingShingle	Procedimento de Fontan Enteropatia por perda de proteínas Septo auricular Cateterismo cardíaco Estenose mitral Dupla saída do ventrículo direito Diseases of the circulatory (Cardiovascular) system RC666-701 Milan Djukic Stefan A. Djordjevic Andrija S. Pavlovic Igor Stefanovic Mirko Topalovic Ingo Dähnert Protein-losing enteropathy managed with percutaneous enlargement of a restrictive atrial septal defect
description	Protein-losing enteropathy is one of the most feared complications of the Fontan circulation. The diagnosis of protein-losing enteropathy in this setting should prompt a thorough investigation for the presence of a treatable hemodynamic impairment. In this report, we describe a complete reversal of protein-losing enteropathy following percutaneous enlargement of a restrictive atrial septal defect in a patient with a fenestrated lateral tunnel Fontan and severe mitral stenosis. Resumo: A enteropatia por perda de proteínas é uma das complicações mais temidas da circulação de Fontan. O diagnóstico de enteropatia por perda de proteínas neste contexto deve suscitar uma avaliação exaustiva da eventual presença de uma alteraçao hemodinâmica tratável. Neste artigo, apresentamos uma completa regressão de enteropatia por perda de proteínas após dilatação percutânea de defeito restritivo do septo interauricular num doente com tunel lateral fenestrado de Fontan e estenose mitral grave.
format	article
author	Milan Djukic Stefan A. Djordjevic Andrija S. Pavlovic Igor Stefanovic Mirko Topalovic Ingo Dähnert
author_facet	Milan Djukic Stefan A. Djordjevic Andrija S. Pavlovic Igor Stefanovic Mirko Topalovic Ingo Dähnert
author_sort	Milan Djukic
title	Protein-losing enteropathy managed with percutaneous enlargement of a restrictive atrial septal defect
title_short	Protein-losing enteropathy managed with percutaneous enlargement of a restrictive atrial septal defect
title_full	Protein-losing enteropathy managed with percutaneous enlargement of a restrictive atrial septal defect
title_fullStr	Protein-losing enteropathy managed with percutaneous enlargement of a restrictive atrial septal defect
title_full_unstemmed	Protein-losing enteropathy managed with percutaneous enlargement of a restrictive atrial septal defect
title_sort	protein-losing enteropathy managed with percutaneous enlargement of a restrictive atrial septal defect
publisher	Elsevier
publishDate	2021
url	https://doaj.org/article/088b12f4e1774c03995d770abcc830dd
work_keys_str_mv	AT milandjukic proteinlosingenteropathymanagedwithpercutaneousenlargementofarestrictiveatrialseptaldefect AT stefanadjordjevic proteinlosingenteropathymanagedwithpercutaneousenlargementofarestrictiveatrialseptaldefect AT andrijaspavlovic proteinlosingenteropathymanagedwithpercutaneousenlargementofarestrictiveatrialseptaldefect AT igorstefanovic proteinlosingenteropathymanagedwithpercutaneousenlargementofarestrictiveatrialseptaldefect AT mirkotopalovic proteinlosingenteropathymanagedwithpercutaneousenlargementofarestrictiveatrialseptaldefect AT ingodahnert proteinlosingenteropathymanagedwithpercutaneousenlargementofarestrictiveatrialseptaldefect
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Protein-losing enteropathy managed with percutaneous enlargement of a restrictive atrial septal defect

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