Anti-phage islands force their target phage to directly mediate island excision and spread

Mobile genetic elements called PLEs protect Vibrio cholerae from infection with phage ICP1 by unclear mechanisms. Here, McKitterick and Seed show that a PLE-encoded large serine recombinase exploits an ICP1 protein as a recombination directionality factor to excise this PLE in response to phage infe...

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Detalles Bibliográficos
Autores principales:	Amelia C. McKitterick, Kimberley D. Seed
Formato:	article
Lenguaje:	EN
Publicado:	Nature Portfolio 2018
Materias:	Science Q
Acceso en línea:	https://doaj.org/article/0e797a9c4ba3467e9bd2c5ec57184a1f
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spelling	oai:doaj.org-article:0e797a9c4ba3467e9bd2c5ec57184a1f2021-12-02T16:56:36ZAnti-phage islands force their target phage to directly mediate island excision and spread10.1038/s41467-018-04786-52041-1723https://doaj.org/article/0e797a9c4ba3467e9bd2c5ec57184a1f2018-06-01T00:00:00Zhttps://doi.org/10.1038/s41467-018-04786-5https://doaj.org/toc/2041-1723Mobile genetic elements called PLEs protect Vibrio cholerae from infection with phage ICP1 by unclear mechanisms. Here, McKitterick and Seed show that a PLE-encoded large serine recombinase exploits an ICP1 protein as a recombination directionality factor to excise this PLE in response to phage infection.Amelia C. McKitterickKimberley D. SeedNature PortfolioarticleScienceQENNature Communications, Vol 9, Iss 1, Pp 1-8 (2018)
institution	DOAJ
collection	DOAJ
language	EN
topic	Science Q
spellingShingle	Science Q Amelia C. McKitterick Kimberley D. Seed Anti-phage islands force their target phage to directly mediate island excision and spread
description	Mobile genetic elements called PLEs protect Vibrio cholerae from infection with phage ICP1 by unclear mechanisms. Here, McKitterick and Seed show that a PLE-encoded large serine recombinase exploits an ICP1 protein as a recombination directionality factor to excise this PLE in response to phage infection.
format	article
author	Amelia C. McKitterick Kimberley D. Seed
author_facet	Amelia C. McKitterick Kimberley D. Seed
author_sort	Amelia C. McKitterick
title	Anti-phage islands force their target phage to directly mediate island excision and spread
title_short	Anti-phage islands force their target phage to directly mediate island excision and spread
title_full	Anti-phage islands force their target phage to directly mediate island excision and spread
title_fullStr	Anti-phage islands force their target phage to directly mediate island excision and spread
title_full_unstemmed	Anti-phage islands force their target phage to directly mediate island excision and spread
title_sort	anti-phage islands force their target phage to directly mediate island excision and spread
publisher	Nature Portfolio
publishDate	2018
url	https://doaj.org/article/0e797a9c4ba3467e9bd2c5ec57184a1f
work_keys_str_mv	AT ameliacmckitterick antiphageislandsforcetheirtargetphagetodirectlymediateislandexcisionandspread AT kimberleydseed antiphageislandsforcetheirtargetphagetodirectlymediateislandexcisionandspread
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Anti-phage islands force their target phage to directly mediate island excision and spread

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