Delaying histone deacetylase response to injury accelerates conversion into repair Schwann cells and nerve regeneration

Brüggeret al. identify part of the molecular machinery that controls Schwann cell development after peripheral nerve injury. Inhibiting HDAC1/2 early after injury enhances nerve regeneration and promotes functional recovery.

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Autores principales:	Valérie Brügger, Mert Duman, Maëlle Bochud, Emmanuelle Münger, Manfred Heller, Sophie Ruff, Claire Jacob
Formato:	article
Lenguaje:	EN
Publicado:	Nature Portfolio 2017
Materias:	Science Q
Acceso en línea:	https://doaj.org/article/1bcc71199b3443bd959d41fc82920f4f
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spelling	oai:doaj.org-article:1bcc71199b3443bd959d41fc82920f4f2021-12-02T15:38:57ZDelaying histone deacetylase response to injury accelerates conversion into repair Schwann cells and nerve regeneration10.1038/ncomms142722041-1723https://doaj.org/article/1bcc71199b3443bd959d41fc82920f4f2017-01-01T00:00:00Zhttps://doi.org/10.1038/ncomms14272https://doaj.org/toc/2041-1723Brüggeret al. identify part of the molecular machinery that controls Schwann cell development after peripheral nerve injury. Inhibiting HDAC1/2 early after injury enhances nerve regeneration and promotes functional recovery.Valérie BrüggerMert DumanMaëlle BochudEmmanuelle MüngerManfred HellerSophie RuffClaire JacobNature PortfolioarticleScienceQENNature Communications, Vol 8, Iss 1, Pp 1-16 (2017)
institution	DOAJ
collection	DOAJ
language	EN
topic	Science Q
spellingShingle	Science Q Valérie Brügger Mert Duman Maëlle Bochud Emmanuelle Münger Manfred Heller Sophie Ruff Claire Jacob Delaying histone deacetylase response to injury accelerates conversion into repair Schwann cells and nerve regeneration
description	Brüggeret al. identify part of the molecular machinery that controls Schwann cell development after peripheral nerve injury. Inhibiting HDAC1/2 early after injury enhances nerve regeneration and promotes functional recovery.
format	article
author	Valérie Brügger Mert Duman Maëlle Bochud Emmanuelle Münger Manfred Heller Sophie Ruff Claire Jacob
author_facet	Valérie Brügger Mert Duman Maëlle Bochud Emmanuelle Münger Manfred Heller Sophie Ruff Claire Jacob
author_sort	Valérie Brügger
title	Delaying histone deacetylase response to injury accelerates conversion into repair Schwann cells and nerve regeneration
title_short	Delaying histone deacetylase response to injury accelerates conversion into repair Schwann cells and nerve regeneration
title_full	Delaying histone deacetylase response to injury accelerates conversion into repair Schwann cells and nerve regeneration
title_fullStr	Delaying histone deacetylase response to injury accelerates conversion into repair Schwann cells and nerve regeneration
title_full_unstemmed	Delaying histone deacetylase response to injury accelerates conversion into repair Schwann cells and nerve regeneration
title_sort	delaying histone deacetylase response to injury accelerates conversion into repair schwann cells and nerve regeneration
publisher	Nature Portfolio
publishDate	2017
url	https://doaj.org/article/1bcc71199b3443bd959d41fc82920f4f
work_keys_str_mv	AT valeriebrugger delayinghistonedeacetylaseresponsetoinjuryacceleratesconversionintorepairschwanncellsandnerveregeneration AT mertduman delayinghistonedeacetylaseresponsetoinjuryacceleratesconversionintorepairschwanncellsandnerveregeneration AT maellebochud delayinghistonedeacetylaseresponsetoinjuryacceleratesconversionintorepairschwanncellsandnerveregeneration AT emmanuellemunger delayinghistonedeacetylaseresponsetoinjuryacceleratesconversionintorepairschwanncellsandnerveregeneration AT manfredheller delayinghistonedeacetylaseresponsetoinjuryacceleratesconversionintorepairschwanncellsandnerveregeneration AT sophieruff delayinghistonedeacetylaseresponsetoinjuryacceleratesconversionintorepairschwanncellsandnerveregeneration AT clairejacob delayinghistonedeacetylaseresponsetoinjuryacceleratesconversionintorepairschwanncellsandnerveregeneration
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Delaying histone deacetylase response to injury accelerates conversion into repair Schwann cells and nerve regeneration

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