Polycystin-1 is required for insulin-like growth factor 1-induced cardiomyocyte hypertrophy.
Cardiac hypertrophy is the result of responses to various physiological or pathological stimuli. Recently, we showed that polycystin-1 participates in cardiomyocyte hypertrophy elicited by pressure overload and mechanical stress. Interestingly, polycystin-1 knockdown does not affect phenylephrine-in...
Guardado en:
Autores principales: | Carolina Fernández, Natalia Torrealba, Francisco Altamirano, Valeria Garrido-Moreno, César Vásquez-Trincado, Raúl Flores-Vergara, Camila López-Crisosto, María Paz Ocaranza, Mario Chiong, Zully Pedrozo, Sergio Lavandero |
---|---|
Formato: | article |
Lenguaje: | EN |
Publicado: |
Public Library of Science (PLoS)
2021
|
Materias: | |
Acceso en línea: | https://doaj.org/article/282852eddbe64e25877af76197d62e17 |
Etiquetas: |
Agregar Etiqueta
Sin Etiquetas, Sea el primero en etiquetar este registro!
|
Ejemplares similares
-
Src is required for mechanical stretch-induced cardiomyocyte hypertrophy through angiotensin II type 1 receptor-dependent β-arrestin2 pathways.
por: Shijun Wang, et al.
Publicado: (2014) -
The Gating of Polycystin Signaling Complex
por: DELMAS,PATRICK
Publicado: (2004) -
Testosterone activates glucose metabolism through AMPK and androgen signaling in cardiomyocyte hypertrophy
por: Troncoso,Mayarling Francisca, et al.
Publicado: (2021) -
Insulin-Induced Cardiomyocytes Hypertrophy That Is Prevented by Taurine via β-alanine-Sensitive Na<sup>+</sup>-Taurine Symporter
por: Ashley Jazzar, et al.
Publicado: (2021) -
DNA hydroxymethylation controls cardiomyocyte gene expression in development and hypertrophy
por: Carolina M. Greco, et al.
Publicado: (2016)