Chemokine CCL2 prevents opioid-induced inhibition of nociceptive synaptic transmission in spinal cord dorsal horn
Abstract Background Opioid analgesics remain widely used for pain treatment despite the related serious side effects. Some of those, such as opioid tolerance and opioid-induced hyperalgesia may be at least partially due to modulation of opioid receptors (OR) function at nociceptive synapses in the s...
Guardado en:
Autores principales: | Mario Heles, Petra Mrozkova, Dominika Sulcova, Pavel Adamek, Diana Spicarova, Jiri Palecek |
---|---|
Formato: | article |
Lenguaje: | EN |
Publicado: |
BMC
2021
|
Materias: | |
Acceso en línea: | https://doaj.org/article/283da2c846314e67b4b0e0c8da04c39f |
Etiquetas: |
Agregar Etiqueta
Sin Etiquetas, Sea el primero en etiquetar este registro!
|
Ejemplares similares
-
CHEMOKINES CCL17 AND CCL22 IN SARCOIDOSIS
por: N. M. Lazareva, et al.
Publicado: (2021) -
The Peripheral Role of CCL2 in the Anti-Nociceptive Effect of Sigma-1 Receptor Antagonist BD1047 on Inflammatory Hyperalgesia in Rats
por: Sungkun Chun, et al.
Publicado: (2021) -
Plasma C-C Chemokine Concentrations in Intermediate Age-Related Macular Degeneration
por: Alan G. Palestine, et al.
Publicado: (2021) -
Itch induced by peripheral mu opioid receptors is dependent on TRPV1-expressing neurons and alleviated by channel activation
por: Helvira Melo, et al.
Publicado: (2018) -
Neuron Type-Dependent Synaptic Activity in the Spinal Dorsal Horn of Opioid-Induced Hyperalgesia Mouse Model
por: Austin Kearns, et al.
Publicado: (2021)