Homocysteine induces mitochondrial dysfunction involving the crosstalk between oxidative stress and mitochondrial pSTAT3 in rat ischemic brain
Abstract Homocysteine (Hcy) has been shown to have a neurotoxic effect on ischemic brain cells; however, the underlying mechanisms remain incompletely understood. Here, we examined whether Hcy treatment influences mitochondria injury, oxidative stress, and mitochondrial STAT3 (mitoStat3) expression...
Guardado en:
Autores principales: | Shuang Chen, Zhiping Dong, Yaqian Zhao, Na Sai, Xuan Wang, Huan Liu, Guowei Huang, Xumei Zhang |
---|---|
Formato: | article |
Lenguaje: | EN |
Publicado: |
Nature Portfolio
2017
|
Materias: | |
Acceso en línea: | https://doaj.org/article/3176e52b40a94194b24290433dbe389c |
Etiquetas: |
Agregar Etiqueta
Sin Etiquetas, Sea el primero en etiquetar este registro!
|
Ejemplares similares
-
Mitochondrial dysfunction in neurological disorders: Exploring mitochondrial transplantation
por: Pedro Norat, et al.
Publicado: (2020) -
Roles of LncRNAs in Regulating Mitochondrial Dysfunction in Septic Cardiomyopathy
por: Shuang Liu, et al.
Publicado: (2021) -
Generation of somatic mitochondrial DNA-replaced cells for mitochondrial dysfunction treatment
por: Hideki Maeda, et al.
Publicado: (2021) -
Author Correction: Mitochondrial dysfunction in neurological disorders: exploring mitochondrial transplantation
por: Pedro Norat, et al.
Publicado: (2021) -
Single organelle analysis to characterize mitochondrial function and crosstalk during viral infection
por: Annika Schneider, et al.
Publicado: (2019)