A molecular mechanism for bacterial susceptibility to zinc.
Transition row metal ions are both essential and toxic to microorganisms. Zinc in excess has significant toxicity to bacteria, and host release of Zn(II) at mucosal surfaces is an important innate defence mechanism. However, the molecular mechanisms by which Zn(II) affords protection have not been d...
Guardado en:
Autores principales: | Christopher A McDevitt, Abiodun D Ogunniyi, Eugene Valkov, Michael C Lawrence, Bostjan Kobe, Alastair G McEwan, James C Paton |
---|---|
Formato: | article |
Lenguaje: | EN |
Publicado: |
Public Library of Science (PLoS)
2011
|
Materias: | |
Acceso en línea: | https://doaj.org/article/328dd368a41b46dca8253adf6a810ac8 |
Etiquetas: |
Agregar Etiqueta
Sin Etiquetas, Sea el primero en etiquetar este registro!
|
Ejemplares similares
-
Extracellular zinc competitively inhibits manganese uptake and compromises oxidative stress management in Streptococcus pneumoniae.
por: Bart A Eijkelkamp, et al.
Publicado: (2014) -
A Trap-Door Mechanism for Zinc Acquisition by
<i>Streptococcus pneumoniae</i>
AdcA
por: Zhenyao Luo, et al.
Publicado: (2021) -
Iwi radio in the era of media convergence: The opportunities and challenges of becoming ‘more than radio.’
por: Rufus McEwan
Publicado: (2019) -
Chemical Synergy between Ionophore PBT2 and Zinc Reverses Antibiotic Resistance
por: Lisa Bohlmann, et al.
Publicado: (2018) -
Molecular basis of regio- and stereo-specificity in biosynthesis of bacterial heterodimeric diketopiperazines
por: Chenghai Sun, et al.
Publicado: (2020)