Selenoprotein DIO2 Is a Regulator of Mitochondrial Function, Morphology and UPRmt in Human Cardiomyocytes

Members of the fetal-gene-program may act as regulatory components to impede deleterious events occurring with cardiac remodeling, and constitute potential novel therapeutic heart failure (HF) targets. Mitochondrial energy derangements occur both during early fetal development and in patients with H...

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Auteurs principaux:	Nils Bomer, Mario G. Pavez-Giani, Frederik E. Deiman, Annet N. Linders, Martijn F. Hoes, Christiane L.J. Baierl, Silke U. Oberdorf-Maass, Rudolf A. de Boer, Herman H.W. Silljé, Eugene Berezikov, Warner S. Simonides, B. Daan Westenbrink, Peter van der Meer
Format:	article
Langue:	EN
Publié:	MDPI AG 2021
Sujets:	heart failure selenoproteins DIO2 human cardiomyocytes mtUPR mitochondrial function Biology (General) QH301-705.5 Chemistry QD1-999
Accès en ligne:	https://doaj.org/article/379f9168c09b4bdca9868e63a97a417b
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Selenoprotein DIO2 Is a Regulator of Mitochondrial Function, Morphology and UPRmt in Human Cardiomyocytes

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