A Virus Hosted in Malaria-Infected Blood Protects against T Cell-Mediated Inflammatory Diseases by Impairing DC Function in a Type I IFN-Dependent Manner

A Virus Hosted in Malaria-Infected Blood Protects against T Cell-Mediated Inflammatory Diseases by Impairing DC Function in a Type I IFN-Dependent Manner

ABSTRACT Coinfections shape immunity and influence the development of inflammatory diseases, resulting in detrimental or beneficial outcome. Coinfections with concurrent Plasmodium species can alter malaria clinical evolution, and malaria infection itself can modulate autoimmune reactions. Yet, the...

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Autores principales: Ali Hassan, Myriam F. Wlodarczyk, Mehdi Benamar, Emilie Bassot, Anna Salvioni, Sahar Kassem, Antoine Berry, Abdelhadi Saoudi, Nicolas Blanchard
Formato: article
Lenguaje:EN
Publicado: American Society for Microbiology 2020
Materias:
CD4 T cell
RNA virus
autoimmunity
coinfection
dendritic cells
inflammation
Microbiology
QR1-502
Acceso en línea:https://doaj.org/article/3a22cb4a960e438f82336675f40a6899
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