GP96 Drives Exacerbation of Secondary Bacterial Pneumonia following Influenza A Virus Infection

Secondary bacterial pneumonia following an influenza A virus (IAV) infection is a major cause of morbidity and mortality. Although it is generally accepted that preceding IAV infection leads to increased susceptibility to secondary bacterial infection, details regarding the pathogenic mechanism dur...

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Autores principales: Tomoko Sumitomo, Masanobu Nakata, Satoshi Nagase, Yuki Takahara, Mariko Honda-Ogawa, Yasushi Mori, Yukako Akamatsu, Masaya Yamaguchi, Shigefumi Okamoto, Shigetada Kawabata
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Publicado: American Society for Microbiology 2021
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Acceso en línea:https://doaj.org/article/42b70f198b2f447bb6155b5cbdd4ad57
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spelling oai:doaj.org-article:42b70f198b2f447bb6155b5cbdd4ad572021-11-03T21:15:04ZGP96 Drives Exacerbation of Secondary Bacterial Pneumonia following Influenza A Virus Infection2150-751110.1128/mBio.03269-20https://doaj.org/article/42b70f198b2f447bb6155b5cbdd4ad572021-06-01T00:00:00Zhttps://journals.asm.org/doi/10.1128/mBio.03269-20https://doaj.org/toc/2150-7511 Secondary bacterial pneumonia following an influenza A virus (IAV) infection is a major cause of morbidity and mortality. Although it is generally accepted that preceding IAV infection leads to increased susceptibility to secondary bacterial infection, details regarding the pathogenic mechanism during the early stage of superinfection remain elusive.Tomoko SumitomoMasanobu NakataSatoshi NagaseYuki TakaharaMariko Honda-OgawaYasushi MoriYukako AkamatsuMasaya YamaguchiShigefumi OkamotoShigetada KawabataAmerican Society for MicrobiologyarticleMicrobiologyQR1-502ENmBio, Vol 12, Iss 3 (2021)
institution DOAJ
collection DOAJ
language EN
topic Microbiology
QR1-502
spellingShingle Microbiology
QR1-502
Tomoko Sumitomo
Masanobu Nakata
Satoshi Nagase
Yuki Takahara
Mariko Honda-Ogawa
Yasushi Mori
Yukako Akamatsu
Masaya Yamaguchi
Shigefumi Okamoto
Shigetada Kawabata
GP96 Drives Exacerbation of Secondary Bacterial Pneumonia following Influenza A Virus Infection
description Secondary bacterial pneumonia following an influenza A virus (IAV) infection is a major cause of morbidity and mortality. Although it is generally accepted that preceding IAV infection leads to increased susceptibility to secondary bacterial infection, details regarding the pathogenic mechanism during the early stage of superinfection remain elusive.
format article
author Tomoko Sumitomo
Masanobu Nakata
Satoshi Nagase
Yuki Takahara
Mariko Honda-Ogawa
Yasushi Mori
Yukako Akamatsu
Masaya Yamaguchi
Shigefumi Okamoto
Shigetada Kawabata
author_facet Tomoko Sumitomo
Masanobu Nakata
Satoshi Nagase
Yuki Takahara
Mariko Honda-Ogawa
Yasushi Mori
Yukako Akamatsu
Masaya Yamaguchi
Shigefumi Okamoto
Shigetada Kawabata
author_sort Tomoko Sumitomo
title GP96 Drives Exacerbation of Secondary Bacterial Pneumonia following Influenza A Virus Infection
title_short GP96 Drives Exacerbation of Secondary Bacterial Pneumonia following Influenza A Virus Infection
title_full GP96 Drives Exacerbation of Secondary Bacterial Pneumonia following Influenza A Virus Infection
title_fullStr GP96 Drives Exacerbation of Secondary Bacterial Pneumonia following Influenza A Virus Infection
title_full_unstemmed GP96 Drives Exacerbation of Secondary Bacterial Pneumonia following Influenza A Virus Infection
title_sort gp96 drives exacerbation of secondary bacterial pneumonia following influenza a virus infection
publisher American Society for Microbiology
publishDate 2021
url https://doaj.org/article/42b70f198b2f447bb6155b5cbdd4ad57
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