Hypertonic stress modulates eNOS function through O-GlcNAc modification at Thr-866

Hypertonic stress modulates eNOS function through O-GlcNAc modification at Thr-866

Abstract O-GlcNAcylation, an energy-sensitive posttranslational modification, can regulate the activity of endothelial nitric oxide synthase (eNOS). Previous studies found that Thr866 is the key site for low-glucose-mediated regulation of eNOS O-GlcNAc. However, it is not known whether this activity...

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Autores principales: Chang Li, An He, Yongzheng Guo, Xiyang Yang, Minghao Luo, Zhe Cheng, Longxiang Huang, Yong Xia, Suxin Luo
Formato: article
Lenguaje:EN
Publicado: Nature Portfolio 2021
Materias:
Medicine
R
Science
Q
Acceso en línea:https://doaj.org/article/43533d2a51c8416b91c95cafcb473062
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