Silencing XIST mitigated lipopolysaccharide (LPS)-induced inflammatory injury in human lung fibroblast WI-38 cells through modulating miR-30b-5p/CCL16 axis and TLR4/NF-κB signaling pathway

Emerging evidence shows that long noncoding RNA (lncRNA) has been a novel insight in various diseases, including pneumonia. Even though lncRNA X-inactive-specific transcript (XIST) is well studied, its role in pneumonia remains to be largely unrevealed.

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Autores principales:	Xu Jiahui, Li Honggui, Lv Ying, Zhang Chang, Chen Yiting, Yu Dezhao
Formato:	article
Lenguaje:	EN
Publicado:	De Gruyter 2021
Materias:	xist mir-30b-5p lps-induced wi-38 cells ccl16 pneumonia tlr4/nf-κb Biology (General) QH301-705.5
Acceso en línea:	https://doaj.org/article/441f2dbd01f84e909052ad1c6142a484
Etiquetas:	Agregar Etiqueta Sin Etiquetas, Sea el primero en etiquetar este registro!

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spelling	oai:doaj.org-article:441f2dbd01f84e909052ad1c6142a4842021-12-05T14:10:40ZSilencing XIST mitigated lipopolysaccharide (LPS)-induced inflammatory injury in human lung fibroblast WI-38 cells through modulating miR-30b-5p/CCL16 axis and TLR4/NF-κB signaling pathway2391-541210.1515/biol-2021-0005https://doaj.org/article/441f2dbd01f84e909052ad1c6142a4842021-02-01T00:00:00Zhttps://doi.org/10.1515/biol-2021-0005https://doaj.org/toc/2391-5412Emerging evidence shows that long noncoding RNA (lncRNA) has been a novel insight in various diseases, including pneumonia. Even though lncRNA X-inactive-specific transcript (XIST) is well studied, its role in pneumonia remains to be largely unrevealed.Xu JiahuiLi HongguiLv YingZhang ChangChen YitingYu DezhaoDe Gruyterarticlexistmir-30b-5plps-induced wi-38 cellsccl16pneumoniatlr4/nf-κbBiology (General)QH301-705.5ENOpen Life Sciences, Vol 16, Iss 1, Pp 108-127 (2021)
institution	DOAJ
collection	DOAJ
language	EN
topic	xist mir-30b-5p lps-induced wi-38 cells ccl16 pneumonia tlr4/nf-κb Biology (General) QH301-705.5
spellingShingle	xist mir-30b-5p lps-induced wi-38 cells ccl16 pneumonia tlr4/nf-κb Biology (General) QH301-705.5 Xu Jiahui Li Honggui Lv Ying Zhang Chang Chen Yiting Yu Dezhao Silencing XIST mitigated lipopolysaccharide (LPS)-induced inflammatory injury in human lung fibroblast WI-38 cells through modulating miR-30b-5p/CCL16 axis and TLR4/NF-κB signaling pathway
description	Emerging evidence shows that long noncoding RNA (lncRNA) has been a novel insight in various diseases, including pneumonia. Even though lncRNA X-inactive-specific transcript (XIST) is well studied, its role in pneumonia remains to be largely unrevealed.
format	article
author	Xu Jiahui Li Honggui Lv Ying Zhang Chang Chen Yiting Yu Dezhao
author_facet	Xu Jiahui Li Honggui Lv Ying Zhang Chang Chen Yiting Yu Dezhao
author_sort	Xu Jiahui
title	Silencing XIST mitigated lipopolysaccharide (LPS)-induced inflammatory injury in human lung fibroblast WI-38 cells through modulating miR-30b-5p/CCL16 axis and TLR4/NF-κB signaling pathway
title_short	Silencing XIST mitigated lipopolysaccharide (LPS)-induced inflammatory injury in human lung fibroblast WI-38 cells through modulating miR-30b-5p/CCL16 axis and TLR4/NF-κB signaling pathway
title_full	Silencing XIST mitigated lipopolysaccharide (LPS)-induced inflammatory injury in human lung fibroblast WI-38 cells through modulating miR-30b-5p/CCL16 axis and TLR4/NF-κB signaling pathway
title_fullStr	Silencing XIST mitigated lipopolysaccharide (LPS)-induced inflammatory injury in human lung fibroblast WI-38 cells through modulating miR-30b-5p/CCL16 axis and TLR4/NF-κB signaling pathway
title_full_unstemmed	Silencing XIST mitigated lipopolysaccharide (LPS)-induced inflammatory injury in human lung fibroblast WI-38 cells through modulating miR-30b-5p/CCL16 axis and TLR4/NF-κB signaling pathway
title_sort	silencing xist mitigated lipopolysaccharide (lps)-induced inflammatory injury in human lung fibroblast wi-38 cells through modulating mir-30b-5p/ccl16 axis and tlr4/nf-κb signaling pathway
publisher	De Gruyter
publishDate	2021
url	https://doaj.org/article/441f2dbd01f84e909052ad1c6142a484
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Silencing XIST mitigated lipopolysaccharide (LPS)-induced inflammatory injury in human lung fibroblast WI-38 cells through modulating miR-30b-5p/CCL16 axis and TLR4/NF-κB signaling pathway

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