Developmental decrease of entorhinal-hippocampal communication in immune-challenged DISC1 knockdown mice
The authors show that mice that mimic the dual genetic-environmental etiology of psychiatric risk have poor lateral entorhinal cortex-dependent recognition memory already at pre-juvenile age and abnormal communication within LECHP-PFC networks throughout development.
Guardado en:
Autores principales: | Xiaxia Xu, Lingzhen Song, Rebecca Kringel, Ileana L. Hanganu-Opatz |
---|---|
Formato: | article |
Lenguaje: | EN |
Publicado: |
Nature Portfolio
2021
|
Materias: | |
Acceso en línea: | https://doaj.org/article/4eeaaffa270c4411b58571707c6c2adb |
Etiquetas: |
Agregar Etiqueta
Sin Etiquetas, Sea el primero en etiquetar este registro!
|
Ejemplares similares
-
Recollection in the human hippocampal-entorhinal cell circuitry
por: Bernhard P. Staresina, et al.
Publicado: (2019) -
Optogenetic dissection of ictal propagation in the hippocampal–entorhinal cortex structures
por: Yi Lu, et al.
Publicado: (2016) -
Correction: Corrigendum: Optogenetic dissection of ictal propagation in the hippocampal–entorhinal cortex structures
por: Yi Lu, et al.
Publicado: (2016) -
CA1 Nampt knockdown recapitulates hippocampal cognitive phenotypes in old mice which nicotinamide mononucleotide improves
por: Sean Johnson, et al.
Publicado: (2018) -
Hippocampal CA1 replay becomes less prominent but more rigid without inputs from medial entorhinal cortex
por: Alireza Chenani, et al.
Publicado: (2019)