Altered social behaviours in neurexin 1α knockout mice resemble core symptoms in neurodevelopmental disorders.
<h4>Background</h4>Copy number variants have emerged as an important genomic cause of common, complex neurodevelopmental disorders. These usually change copy number of multiple genes, but deletions at 2p16.3, which have been associated with autism, schizophrenia and mental retardation, a...
Guardado en:
Autores principales: | Hannah Mary Grayton, Markus Missler, David Andrew Collier, Cathy Fernandes |
---|---|
Formato: | article |
Lenguaje: | EN |
Publicado: |
Public Library of Science (PLoS)
2013
|
Materias: | |
Acceso en línea: | https://doaj.org/article/572a5183b1af4c6f8167db5aec08bb69 |
Etiquetas: |
Agregar Etiqueta
Sin Etiquetas, Sea el primero en etiquetar este registro!
|
Ejemplares similares
-
IgSF21 promotes differentiation of inhibitory synapses via binding to neurexin2α
por: Yuko Tanabe, et al.
Publicado: (2017) -
Neurexins regulate presynaptic GABAB-receptors at central synapses
por: Fujun Luo, et al.
Publicado: (2021) -
Presenilin/γ-secretase regulates neurexin processing at synapses.
por: Carlos A Saura, et al.
Publicado: (2011) -
Structural insights into modulation and selectivity of transsynaptic neurexin–LRRTM interaction
por: Atsushi Yamagata, et al.
Publicado: (2018) -
Brain specific Lamellipodin knockout results in hyperactivity and increased anxiety of mice
por: Cristian Bodo, et al.
Publicado: (2017)