SCL/TAL1 cooperates with Polycomb RYBP-PRC1 to suppress alternative lineages in blood-fated cells

Mechanisms that operate during embryonic development to restrict cell fate are currently under investigation. Here the authors characterise the role of SCL/TAL1 at the onset of blood specification in embryonic development using mouse EB differentiation culture as a model system.

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Autores principales:	Hedia Chagraoui, Maiken S. Kristiansen, Juan Pablo Ruiz, Ana Serra-Barros, Johanna Richter, Elisa Hall-Ponselé, Nicki Gray, Dominic Waithe, Kevin Clark, Philip Hublitz, Emmanouela Repapi, Georg Otto, Paul Sopp, Stephen Taylor, Supat Thongjuea, Paresh Vyas, Catherine Porcher
Formato:	article
Lenguaje:	EN
Publicado:	Nature Portfolio 2018
Materias:	Science Q
Acceso en línea:	https://doaj.org/article/58afad89e66d460dbd6feaaa3566ef20
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spelling	oai:doaj.org-article:58afad89e66d460dbd6feaaa3566ef202021-12-02T15:34:23ZSCL/TAL1 cooperates with Polycomb RYBP-PRC1 to suppress alternative lineages in blood-fated cells10.1038/s41467-018-07787-62041-1723https://doaj.org/article/58afad89e66d460dbd6feaaa3566ef202018-12-01T00:00:00Zhttps://doi.org/10.1038/s41467-018-07787-6https://doaj.org/toc/2041-1723Mechanisms that operate during embryonic development to restrict cell fate are currently under investigation. Here the authors characterise the role of SCL/TAL1 at the onset of blood specification in embryonic development using mouse EB differentiation culture as a model system.Hedia ChagraouiMaiken S. KristiansenJuan Pablo RuizAna Serra-BarrosJohanna RichterElisa Hall-PonseléNicki GrayDominic WaitheKevin ClarkPhilip HublitzEmmanouela RepapiGeorg OttoPaul SoppStephen TaylorSupat ThongjueaParesh VyasCatherine PorcherNature PortfolioarticleScienceQENNature Communications, Vol 9, Iss 1, Pp 1-17 (2018)
institution	DOAJ
collection	DOAJ
language	EN
topic	Science Q
spellingShingle	Science Q Hedia Chagraoui Maiken S. Kristiansen Juan Pablo Ruiz Ana Serra-Barros Johanna Richter Elisa Hall-Ponselé Nicki Gray Dominic Waithe Kevin Clark Philip Hublitz Emmanouela Repapi Georg Otto Paul Sopp Stephen Taylor Supat Thongjuea Paresh Vyas Catherine Porcher SCL/TAL1 cooperates with Polycomb RYBP-PRC1 to suppress alternative lineages in blood-fated cells
description	Mechanisms that operate during embryonic development to restrict cell fate are currently under investigation. Here the authors characterise the role of SCL/TAL1 at the onset of blood specification in embryonic development using mouse EB differentiation culture as a model system.
format	article
author	Hedia Chagraoui Maiken S. Kristiansen Juan Pablo Ruiz Ana Serra-Barros Johanna Richter Elisa Hall-Ponselé Nicki Gray Dominic Waithe Kevin Clark Philip Hublitz Emmanouela Repapi Georg Otto Paul Sopp Stephen Taylor Supat Thongjuea Paresh Vyas Catherine Porcher
author_facet	Hedia Chagraoui Maiken S. Kristiansen Juan Pablo Ruiz Ana Serra-Barros Johanna Richter Elisa Hall-Ponselé Nicki Gray Dominic Waithe Kevin Clark Philip Hublitz Emmanouela Repapi Georg Otto Paul Sopp Stephen Taylor Supat Thongjuea Paresh Vyas Catherine Porcher
author_sort	Hedia Chagraoui
title	SCL/TAL1 cooperates with Polycomb RYBP-PRC1 to suppress alternative lineages in blood-fated cells
title_short	SCL/TAL1 cooperates with Polycomb RYBP-PRC1 to suppress alternative lineages in blood-fated cells
title_full	SCL/TAL1 cooperates with Polycomb RYBP-PRC1 to suppress alternative lineages in blood-fated cells
title_fullStr	SCL/TAL1 cooperates with Polycomb RYBP-PRC1 to suppress alternative lineages in blood-fated cells
title_full_unstemmed	SCL/TAL1 cooperates with Polycomb RYBP-PRC1 to suppress alternative lineages in blood-fated cells
title_sort	scl/tal1 cooperates with polycomb rybp-prc1 to suppress alternative lineages in blood-fated cells
publisher	Nature Portfolio
publishDate	2018
url	https://doaj.org/article/58afad89e66d460dbd6feaaa3566ef20
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SCL/TAL1 cooperates with Polycomb RYBP-PRC1 to suppress alternative lineages in blood-fated cells

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