Down-regulation of neogenin accelerated glioma progression through promoter Methylation and its overexpression in SHG-44 Induced Apoptosis.
<h4>Background</h4>Dependence receptors have been proved to act as tumor suppressors in tumorigenesis. Neogenin, a DCC homologue, well known for its fundamental role in axon guidance and cellular differentiation, is also a dependence receptor functioning to control apoptosis. However, lo...
Guardado en:
Autores principales: | Xinmin Wu, Yunqian Li, Xilin Wan, Tabitha Mlowoka Kayira, Rangjuan Cao, Xingda Ju, Xiaojuan Zhu, Gang Zhao |
---|---|
Formato: | article |
Lenguaje: | EN |
Publicado: |
Public Library of Science (PLoS)
2012
|
Materias: | |
Acceso en línea: | https://doaj.org/article/5c7692f269354978a99bee31edab2ebc |
Etiquetas: |
Agregar Etiqueta
Sin Etiquetas, Sea el primero en etiquetar este registro!
|
Ejemplares similares
-
A systematic study of PPLN length dependence in intracavity SHG
por: Tyler Kashak, et al.
Publicado: (2021) -
Neogenin recruitment of the WAVE regulatory complex maintains adherens junction stability and tension
por: Natalie K. Lee, et al.
Publicado: (2016) -
Ephrin-A5 potentiates netrin-1 axon guidance by enhancing Neogenin availability
por: L.-P. Croteau, et al.
Publicado: (2019) -
Niche derived netrin-1 regulates hematopoietic stem cell dormancy via its receptor neogenin-1
por: Simon Renders, et al.
Publicado: (2021) -
Glioma-derived IL-33 orchestrates an inflammatory brain tumor microenvironment that accelerates glioma progression
por: Astrid De Boeck, et al.
Publicado: (2020)