A miR-150/TET3 pathway regulates the generation of mouse and human non-classical monocyte subset
A decrease in the fraction of non-classical monocytes is a hallmark of chronic myelomonocytic leukaemia. Taking advantage of this abnormal situation, the authors identify a mechanistic link between miR-150 and TET3 as being involved in monocyte subset generation.
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Nature Portfolio
2018
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oai:doaj.org-article:5d55187610e84c0189ca6173abd8c67a2021-12-02T14:41:17ZA miR-150/TET3 pathway regulates the generation of mouse and human non-classical monocyte subset10.1038/s41467-018-07801-x2041-1723https://doaj.org/article/5d55187610e84c0189ca6173abd8c67a2018-12-01T00:00:00Zhttps://doi.org/10.1038/s41467-018-07801-xhttps://doaj.org/toc/2041-1723A decrease in the fraction of non-classical monocytes is a hallmark of chronic myelomonocytic leukaemia. Taking advantage of this abnormal situation, the authors identify a mechanistic link between miR-150 and TET3 as being involved in monocyte subset generation.Dorothée Selimoglu-BuetJulie RivièreHussein GhamlouchLaura BencheikhCatherine LacoutMargot MorabitoM’boyba DiopGuillaume MeuriceMarie BrecklerAurélie ChauveauCamille DebordFranck DebeurmeRaphael ItzyksonNicolas ChapuisChristophe WillekensOrianne Wagner-BallonOlivier A. BernardNathalie DroinEric SolaryNature PortfolioarticleScienceQENNature Communications, Vol 9, Iss 1, Pp 1-17 (2018) |
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DOAJ |
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DOAJ |
language |
EN |
topic |
Science Q |
spellingShingle |
Science Q Dorothée Selimoglu-Buet Julie Rivière Hussein Ghamlouch Laura Bencheikh Catherine Lacout Margot Morabito M’boyba Diop Guillaume Meurice Marie Breckler Aurélie Chauveau Camille Debord Franck Debeurme Raphael Itzykson Nicolas Chapuis Christophe Willekens Orianne Wagner-Ballon Olivier A. Bernard Nathalie Droin Eric Solary A miR-150/TET3 pathway regulates the generation of mouse and human non-classical monocyte subset |
description |
A decrease in the fraction of non-classical monocytes is a hallmark of chronic myelomonocytic leukaemia. Taking advantage of this abnormal situation, the authors identify a mechanistic link between miR-150 and TET3 as being involved in monocyte subset generation. |
format |
article |
author |
Dorothée Selimoglu-Buet Julie Rivière Hussein Ghamlouch Laura Bencheikh Catherine Lacout Margot Morabito M’boyba Diop Guillaume Meurice Marie Breckler Aurélie Chauveau Camille Debord Franck Debeurme Raphael Itzykson Nicolas Chapuis Christophe Willekens Orianne Wagner-Ballon Olivier A. Bernard Nathalie Droin Eric Solary |
author_facet |
Dorothée Selimoglu-Buet Julie Rivière Hussein Ghamlouch Laura Bencheikh Catherine Lacout Margot Morabito M’boyba Diop Guillaume Meurice Marie Breckler Aurélie Chauveau Camille Debord Franck Debeurme Raphael Itzykson Nicolas Chapuis Christophe Willekens Orianne Wagner-Ballon Olivier A. Bernard Nathalie Droin Eric Solary |
author_sort |
Dorothée Selimoglu-Buet |
title |
A miR-150/TET3 pathway regulates the generation of mouse and human non-classical monocyte subset |
title_short |
A miR-150/TET3 pathway regulates the generation of mouse and human non-classical monocyte subset |
title_full |
A miR-150/TET3 pathway regulates the generation of mouse and human non-classical monocyte subset |
title_fullStr |
A miR-150/TET3 pathway regulates the generation of mouse and human non-classical monocyte subset |
title_full_unstemmed |
A miR-150/TET3 pathway regulates the generation of mouse and human non-classical monocyte subset |
title_sort |
mir-150/tet3 pathway regulates the generation of mouse and human non-classical monocyte subset |
publisher |
Nature Portfolio |
publishDate |
2018 |
url |
https://doaj.org/article/5d55187610e84c0189ca6173abd8c67a |
work_keys_str_mv |
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