$Wild type transthyretin cardiac amiloidozis as a rare and overlooked underlying etiology in a patient with heart failure with preserved ejection fraction and left ventricular hypertrophy$
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Wild type transthyretin cardiac amiloidozis as a rare and overlooked underlying etiology in a patient with heart failure with preserved ejection fraction and left ventricular hypertrophy

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Autores principales:	Selda Murat, Yüksel Çavuşoğlu, İlknur Ak Sivrikoz
Formato:	article
Lenguaje:	EN TR
Publicado:	KARE Publishing 2021
Materias:	Medicine R Internal medicine RC31-1245 Diseases of the circulatory (Cardiovascular) system RC666-701
Acceso en línea:	https://doaj.org/article/61c3c757fb7849c1a3255bfe3be20ce9
Etiquetas:	Agregar Etiqueta Sin Etiquetas, Sea el primero en etiquetar este registro!

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spelling	oai:doaj.org-article:61c3c757fb7849c1a3255bfe3be20ce92021-11-08T21:03:51ZWild type transthyretin cardiac amiloidozis as a rare and overlooked underlying etiology in a patient with heart failure with preserved ejection fraction and left ventricular hypertrophy10.5543/tkda.2021.093101016-5169https://doaj.org/article/61c3c757fb7849c1a3255bfe3be20ce92021-10-01T00:00:00Zhttps://archivestsc.com/jvi.aspx?un=TKDA-09310&volume=49&issue=7https://doaj.org/toc/1016-5169Selda MuratYüksel Çavuşoğluİlknur Ak SivrikozKARE PublishingarticleMedicineRInternal medicineRC31-1245Diseases of the circulatory (Cardiovascular) systemRC666-701ENTRTürk Kardiyoloji Derneği Arşivi, Vol 49, Iss 7, Pp 579-584 (2021)
institution	DOAJ
collection	DOAJ
language	EN TR
topic	Medicine R Internal medicine RC31-1245 Diseases of the circulatory (Cardiovascular) system RC666-701
spellingShingle	Medicine R Internal medicine RC31-1245 Diseases of the circulatory (Cardiovascular) system RC666-701 Selda Murat Yüksel Çavuşoğlu İlknur Ak Sivrikoz Wild type transthyretin cardiac amiloidozis as a rare and overlooked underlying etiology in a patient with heart failure with preserved ejection fraction and left ventricular hypertrophy
format	article
author	Selda Murat Yüksel Çavuşoğlu İlknur Ak Sivrikoz
author_facet	Selda Murat Yüksel Çavuşoğlu İlknur Ak Sivrikoz
author_sort	Selda Murat
title	Wild type transthyretin cardiac amiloidozis as a rare and overlooked underlying etiology in a patient with heart failure with preserved ejection fraction and left ventricular hypertrophy
title_short	Wild type transthyretin cardiac amiloidozis as a rare and overlooked underlying etiology in a patient with heart failure with preserved ejection fraction and left ventricular hypertrophy
title_full	Wild type transthyretin cardiac amiloidozis as a rare and overlooked underlying etiology in a patient with heart failure with preserved ejection fraction and left ventricular hypertrophy
title_fullStr	Wild type transthyretin cardiac amiloidozis as a rare and overlooked underlying etiology in a patient with heart failure with preserved ejection fraction and left ventricular hypertrophy
title_full_unstemmed	Wild type transthyretin cardiac amiloidozis as a rare and overlooked underlying etiology in a patient with heart failure with preserved ejection fraction and left ventricular hypertrophy
title_sort	wild type transthyretin cardiac amiloidozis as a rare and overlooked underlying etiology in a patient with heart failure with preserved ejection fraction and left ventricular hypertrophy
publisher	KARE Publishing
publishDate	2021
url	https://doaj.org/article/61c3c757fb7849c1a3255bfe3be20ce9
work_keys_str_mv	AT seldamurat wildtypetransthyretincardiacamiloidozisasarareandoverlookedunderlyingetiologyinapatientwithheartfailurewithpreservedejectionfractionandleftventricularhypertrophy AT yukselcavusoglu wildtypetransthyretincardiacamiloidozisasarareandoverlookedunderlyingetiologyinapatientwithheartfailurewithpreservedejectionfractionandleftventricularhypertrophy AT ilknuraksivrikoz wildtypetransthyretincardiacamiloidozisasarareandoverlookedunderlyingetiologyinapatientwithheartfailurewithpreservedejectionfractionandleftventricularhypertrophy
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Wild type transthyretin cardiac amiloidozis as a rare and overlooked underlying etiology in a patient with heart failure with preserved ejection fraction and left ventricular hypertrophy

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