Mitochondria-specific drug release and reactive oxygen species burst induced by polyprodrug nanoreactors can enhance chemotherapy
Mitochondria are a source of reactive oxygen species, which can be exploited to induce the death of cancer cells. Here, the authors use nanoparticles that release camptothecin in a reactive oxygen species dependent manner, leading to cancer cell death.
Guardado en:
Autores principales: | Wenjia Zhang, Xianglong Hu, Qi Shen, Da Xing |
---|---|
Formato: | article |
Lenguaje: | EN |
Publicado: |
Nature Portfolio
2019
|
Materias: | |
Acceso en línea: | https://doaj.org/article/6d5da74a11b647ee870c25a0246705d3 |
Etiquetas: |
Agregar Etiqueta
Sin Etiquetas, Sea el primero en etiquetar este registro!
|
Ejemplares similares
-
Author Correction: Mitochondria-specific drug release and reactive oxygen species burst induced by polyprodrug nanoreactors can enhance chemotherapy
por: Wenjia Zhang, et al.
Publicado: (2019) -
Author Correction: Mitochondria-specific drug release and reactive oxygen species burst induced by polyprodrug nanoreactors can enhance chemotherapy
por: Wenjia Zhang, et al.
Publicado: (2019) -
Targeted production of reactive oxygen species in mitochondria to overcome cancer drug resistance
por: Hai Wang, et al.
Publicado: (2018) -
Arsenene-mediated multiple independently targeted reactive oxygen species burst for cancer therapy
por: Na Kong, et al.
Publicado: (2021) -
Mitochondria-Shaping Proteins and Chemotherapy
por: Longlong Xie, et al.
Publicado: (2021)