Cigarette smoke modulates expression of human rhinovirus-induced airway epithelial host defense genes.
Human rhinovirus (HRV) infections trigger acute exacerbations of chronic obstructive pulmonary disease (COPD) and asthma. The human airway epithelial cell is the primary site of HRV infection and responds to infection with altered expression of multiple genes, the products of which could regulate th...
Guardado en:
Autores principales: | David Proud, Magdalena H Hudy, Shahina Wiehler, Raza S Zaheer, Minaa A Amin, Jonathan B Pelikan, Claire E Tacon, Tabitha O Tonsaker, Brandie L Walker, Cora Kooi, Suzanne L Traves, Richard Leigh |
---|---|
Formato: | article |
Lenguaje: | EN |
Publicado: |
Public Library of Science (PLoS)
2012
|
Materias: | |
Acceso en línea: | https://doaj.org/article/7d437a8870424a809de34846b6cb4dea |
Etiquetas: |
Agregar Etiqueta
Sin Etiquetas, Sea el primero en etiquetar este registro!
|
Ejemplares similares
-
PGC-1α mediates a metabolic host defense response in human airway epithelium during rhinovirus infections
por: Aubrey N. Michi, et al.
Publicado: (2021) -
Characterising the mechanism of airway smooth muscle β2 adrenoceptor desensitization by rhinovirus infected bronchial epithelial cells.
por: David Van Ly, et al.
Publicado: (2013) -
Mechanism of Rhinovirus Immunity and Asthma
por: Zuqin Yang, et al.
Publicado: (2021) -
Population structure and evolution of Rhinoviruses.
por: Vaishali P Waman, et al.
Publicado: (2014) -
A Small Molecule BH3-mimetic Suppresses Cigarette Smoke-Induced Mucous Expression in Airway Epithelial Cells
por: Shah S. Hussain, et al.
Publicado: (2018)