NMI and IFP35 serve as proinflammatory DAMPs during cellular infection and injury

Damage-associated molecular patterns (DAMP) are important mediators of innate immunity. Here the authors show that N-myc and STAT interactor (NMI) and interferon-induced protein 35 (IFP35) act as DAMPs to promote inflammation by activating macrophages via the Toll-like receptor 4 and NF-κB pathways....

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Autores principales:	Zhikai Xiahou, Xiangli Wang, Juan Shen, Xiaoxiao Zhu, Feng Xu, Rong Hu, Deyin Guo, Henan Li, Yong Tian, Yingfang Liu, Huanhuan Liang
Formato:	article
Lenguaje:	EN
Publicado:	Nature Portfolio 2017
Materias:	Science Q
Acceso en línea:	https://doaj.org/article/85dba436a8234d6ba7c372cf2c091ff3
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spelling	oai:doaj.org-article:85dba436a8234d6ba7c372cf2c091ff32021-12-02T14:40:24ZNMI and IFP35 serve as proinflammatory DAMPs during cellular infection and injury10.1038/s41467-017-00930-92041-1723https://doaj.org/article/85dba436a8234d6ba7c372cf2c091ff32017-10-01T00:00:00Zhttps://doi.org/10.1038/s41467-017-00930-9https://doaj.org/toc/2041-1723Damage-associated molecular patterns (DAMP) are important mediators of innate immunity. Here the authors show that N-myc and STAT interactor (NMI) and interferon-induced protein 35 (IFP35) act as DAMPs to promote inflammation by activating macrophages via the Toll-like receptor 4 and NF-κB pathways.Zhikai XiahouXiangli WangJuan ShenXiaoxiao ZhuFeng XuRong HuDeyin GuoHenan LiYong TianYingfang LiuHuanhuan LiangNature PortfolioarticleScienceQENNature Communications, Vol 8, Iss 1, Pp 1-11 (2017)
institution	DOAJ
collection	DOAJ
language	EN
topic	Science Q
spellingShingle	Science Q Zhikai Xiahou Xiangli Wang Juan Shen Xiaoxiao Zhu Feng Xu Rong Hu Deyin Guo Henan Li Yong Tian Yingfang Liu Huanhuan Liang NMI and IFP35 serve as proinflammatory DAMPs during cellular infection and injury
description	Damage-associated molecular patterns (DAMP) are important mediators of innate immunity. Here the authors show that N-myc and STAT interactor (NMI) and interferon-induced protein 35 (IFP35) act as DAMPs to promote inflammation by activating macrophages via the Toll-like receptor 4 and NF-κB pathways.
format	article
author	Zhikai Xiahou Xiangli Wang Juan Shen Xiaoxiao Zhu Feng Xu Rong Hu Deyin Guo Henan Li Yong Tian Yingfang Liu Huanhuan Liang
author_facet	Zhikai Xiahou Xiangli Wang Juan Shen Xiaoxiao Zhu Feng Xu Rong Hu Deyin Guo Henan Li Yong Tian Yingfang Liu Huanhuan Liang
author_sort	Zhikai Xiahou
title	NMI and IFP35 serve as proinflammatory DAMPs during cellular infection and injury
title_short	NMI and IFP35 serve as proinflammatory DAMPs during cellular infection and injury
title_full	NMI and IFP35 serve as proinflammatory DAMPs during cellular infection and injury
title_fullStr	NMI and IFP35 serve as proinflammatory DAMPs during cellular infection and injury
title_full_unstemmed	NMI and IFP35 serve as proinflammatory DAMPs during cellular infection and injury
title_sort	nmi and ifp35 serve as proinflammatory damps during cellular infection and injury
publisher	Nature Portfolio
publishDate	2017
url	https://doaj.org/article/85dba436a8234d6ba7c372cf2c091ff3
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_version_	1718390307430596608

NMI and IFP35 serve as proinflammatory DAMPs during cellular infection and injury

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