In-host evolution of Staphylococcus epidermidis in a pacemaker-associated endocarditis resulting in increased antibiotic tolerance

Staphylococcus epidermidis is a frequent cause of medical implant-associated biofilm infections. Here, studying a patient with pacemaker-associated endocarditis, the authors report in-host evolution of S. epidermidis leading to phenotypes exhibiting increased biofilm formation and antibiotic toleran...

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Autores principales:	Vanina Dengler Haunreiter, Mathilde Boumasmoud, Nicola Häffner, Dennis Wipfli, Nadja Leimer, Carole Rachmühl, Denise Kühnert, Yvonne Achermann, Reinhard Zbinden, Stefano Benussi, Clement Vulin, Annelies S. Zinkernagel
Formato:	article
Lenguaje:	EN
Publicado:	Nature Portfolio 2019
Materias:	Science Q
Acceso en línea:	https://doaj.org/article/8fa482bb0c6a4cc980cfed4350010340
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spelling	oai:doaj.org-article:8fa482bb0c6a4cc980cfed43500103402021-12-02T15:36:03ZIn-host evolution of Staphylococcus epidermidis in a pacemaker-associated endocarditis resulting in increased antibiotic tolerance10.1038/s41467-019-09053-92041-1723https://doaj.org/article/8fa482bb0c6a4cc980cfed43500103402019-03-01T00:00:00Zhttps://doi.org/10.1038/s41467-019-09053-9https://doaj.org/toc/2041-1723Staphylococcus epidermidis is a frequent cause of medical implant-associated biofilm infections. Here, studying a patient with pacemaker-associated endocarditis, the authors report in-host evolution of S. epidermidis leading to phenotypes exhibiting increased biofilm formation and antibiotic tolerance.Vanina Dengler HaunreiterMathilde BoumasmoudNicola HäffnerDennis WipfliNadja LeimerCarole RachmühlDenise KühnertYvonne AchermannReinhard ZbindenStefano BenussiClement VulinAnnelies S. ZinkernagelNature PortfolioarticleScienceQENNature Communications, Vol 10, Iss 1, Pp 1-14 (2019)
institution	DOAJ
collection	DOAJ
language	EN
topic	Science Q
spellingShingle	Science Q Vanina Dengler Haunreiter Mathilde Boumasmoud Nicola Häffner Dennis Wipfli Nadja Leimer Carole Rachmühl Denise Kühnert Yvonne Achermann Reinhard Zbinden Stefano Benussi Clement Vulin Annelies S. Zinkernagel In-host evolution of Staphylococcus epidermidis in a pacemaker-associated endocarditis resulting in increased antibiotic tolerance
description	Staphylococcus epidermidis is a frequent cause of medical implant-associated biofilm infections. Here, studying a patient with pacemaker-associated endocarditis, the authors report in-host evolution of S. epidermidis leading to phenotypes exhibiting increased biofilm formation and antibiotic tolerance.
format	article
author	Vanina Dengler Haunreiter Mathilde Boumasmoud Nicola Häffner Dennis Wipfli Nadja Leimer Carole Rachmühl Denise Kühnert Yvonne Achermann Reinhard Zbinden Stefano Benussi Clement Vulin Annelies S. Zinkernagel
author_facet	Vanina Dengler Haunreiter Mathilde Boumasmoud Nicola Häffner Dennis Wipfli Nadja Leimer Carole Rachmühl Denise Kühnert Yvonne Achermann Reinhard Zbinden Stefano Benussi Clement Vulin Annelies S. Zinkernagel
author_sort	Vanina Dengler Haunreiter
title	In-host evolution of Staphylococcus epidermidis in a pacemaker-associated endocarditis resulting in increased antibiotic tolerance
title_short	In-host evolution of Staphylococcus epidermidis in a pacemaker-associated endocarditis resulting in increased antibiotic tolerance
title_full	In-host evolution of Staphylococcus epidermidis in a pacemaker-associated endocarditis resulting in increased antibiotic tolerance
title_fullStr	In-host evolution of Staphylococcus epidermidis in a pacemaker-associated endocarditis resulting in increased antibiotic tolerance
title_full_unstemmed	In-host evolution of Staphylococcus epidermidis in a pacemaker-associated endocarditis resulting in increased antibiotic tolerance
title_sort	in-host evolution of staphylococcus epidermidis in a pacemaker-associated endocarditis resulting in increased antibiotic tolerance
publisher	Nature Portfolio
publishDate	2019
url	https://doaj.org/article/8fa482bb0c6a4cc980cfed4350010340
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In-host evolution of Staphylococcus epidermidis in a pacemaker-associated endocarditis resulting in increased antibiotic tolerance

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