Host surface ectonucleotidase-CD73 and the opportunistic pathogen, Porphyromonas gingivalis, cross-modulation underlies a new homeostatic mechanism for chronic bacterial survival in human epithelial cells
Cell surface nucleotide-metabolizing enzyme, ectonucleotidase-CD73, has emerged as a central component of the cellular homeostatic-machinery that counterbalances the danger-molecule (extracellular-ATP)-driven proinflammatory response in immune cells. While the importance of CD73 in microbial host fi...
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Autores principales: | Jaden S. Lee, Nityananda Chowdhury, JoAnn S. Roberts, Özlem Yilmaz |
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Formato: | article |
Lenguaje: | EN |
Publicado: |
Taylor & Francis Group
2020
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Materias: | |
Acceso en línea: | https://doaj.org/article/9e982bd611154620955d119b2d02c9bc |
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