Oyster-Derived Tyr-Ala (YA) Peptide Prevents Lipopolysaccharide/D-Galactosamine-Induced Acute Liver Failure by Suppressing Inflammatory, Apoptotic, Ferroptotic, and Pyroptotic Signals

Oyster-Derived Tyr-Ala (YA) Peptide Prevents Lipopolysaccharide/D-Galactosamine-Induced Acute Liver Failure by Suppressing Inflammatory, Apoptotic, Ferroptotic, and Pyroptotic Signals

Models created by the intraperitoneal injection of lipopolysaccharide (LPS) and D-galactosamine (D-GalN) have been widely used to study the pathogenesis of human acute liver failure (ALF) and drug development. Our previous study reported that oyster (Crassostrea gigas) hydrolysate (OH) had a hepatop...

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Autores principales: Adrian S. Siregar, Marie Merci Nyiramana, Eun-Jin Kim, Soo Buem Cho, Min Seok Woo, Dong Kun Lee, Seong-Geun Hong, Jaehee Han, Sang Soo Kang, Deok Ryong Kim, Yeung Joon Choi, Dawon Kang
Formato: article
Lenguaje:EN
Publicado: MDPI AG 2021
Materias:
acute liver injury
apoptosis
ferroptosis
inflammation
oyster
peptide
Biology (General)
QH301-705.5
Acceso en línea:https://doaj.org/article/a748721d4c68402dbc1cf018aac77e11
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