Level of membrane-bound hemoglobin and red-cell membrane proteins in patients with essential hypertension complicated and not complicated with metabolic syndrome

We studied the effect of different levels of membrane-bound hemoglobin on the level of red-cell membrane proteins and also their interrelation in patients with essential hypertension with and without metabolic syndrome. It wasfound that high membrane-bound hemoglobin is closely related to the low le...

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Autores principales: Y. I. Pivovarov, E. E. Kuznetsova, V. G. Gorokhova, A. S. Sergeyeva, I. V. Babushkina, L. B. Koryakina, E. O. Andreyeva
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Lenguaje:RU
Publicado: Scientific Сentre for Family Health and Human Reproduction Problems 2016
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Acceso en línea:https://doaj.org/article/aa8ea091c34c41d48206995fadc3dc14
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spelling oai:doaj.org-article:aa8ea091c34c41d48206995fadc3dc142021-11-23T06:14:35ZLevel of membrane-bound hemoglobin and red-cell membrane proteins in patients with essential hypertension complicated and not complicated with metabolic syndrome2541-94202587-959610.12737/22960https://doaj.org/article/aa8ea091c34c41d48206995fadc3dc142016-07-01T00:00:00Zhttps://www.actabiomedica.ru/jour/article/view/243https://doaj.org/toc/2541-9420https://doaj.org/toc/2587-9596We studied the effect of different levels of membrane-bound hemoglobin on the level of red-cell membrane proteins and also their interrelation in patients with essential hypertension with and without metabolic syndrome. It wasfound that high membrane-bound hemoglobin is closely related to the low level of high-density lipoproteins and high level of low-density lipoproteins in patients with essential hypertension complicated with metabolic syndrome. In patients with essential hypertension not complicated with metabolic syndrome high membrane-bound hemoglobin is related to the increased prothrombin time and decreased blood urea nitrogen. In patients with essential hypertension complicated with metabolic syndrome high membrane-bound hemoglobin significantly influences the level of membrane contractile proteins (actin, tropomiosine). In patients with essential hypertension without metabolic syndrome high membrane-bound hemoglobin is accompanied by the decrease of structural and integral membrane proteins levels (anion-transport protein and protein 4.1). As the result of quantitative changes in these proteins and change in their interrelations in patients with ssential hypertension complicated with metabolic syndrome more intensive disorders of structural and functional organization of red-cell membrane can appear.Y. I. PivovarovE. E. KuznetsovaV. G. GorokhovaA. S. SergeyevaI. V. BabushkinaL. B. KoryakinaE. O. AndreyevaScientific Сentre for Family Health and Human Reproduction Problemsarticleessential hypertensionred-cell membrane proteinsmembrane-bound hemoglobinmetabolic syndromeScienceQRUActa Biomedica Scientifica, Vol 1, Iss 4, Pp 61-67 (2016)
institution DOAJ
collection DOAJ
language RU
topic essential hypertension
red-cell membrane proteins
membrane-bound hemoglobin
metabolic syndrome
Science
Q
spellingShingle essential hypertension
red-cell membrane proteins
membrane-bound hemoglobin
metabolic syndrome
Science
Q
Y. I. Pivovarov
E. E. Kuznetsova
V. G. Gorokhova
A. S. Sergeyeva
I. V. Babushkina
L. B. Koryakina
E. O. Andreyeva
Level of membrane-bound hemoglobin and red-cell membrane proteins in patients with essential hypertension complicated and not complicated with metabolic syndrome
description We studied the effect of different levels of membrane-bound hemoglobin on the level of red-cell membrane proteins and also their interrelation in patients with essential hypertension with and without metabolic syndrome. It wasfound that high membrane-bound hemoglobin is closely related to the low level of high-density lipoproteins and high level of low-density lipoproteins in patients with essential hypertension complicated with metabolic syndrome. In patients with essential hypertension not complicated with metabolic syndrome high membrane-bound hemoglobin is related to the increased prothrombin time and decreased blood urea nitrogen. In patients with essential hypertension complicated with metabolic syndrome high membrane-bound hemoglobin significantly influences the level of membrane contractile proteins (actin, tropomiosine). In patients with essential hypertension without metabolic syndrome high membrane-bound hemoglobin is accompanied by the decrease of structural and integral membrane proteins levels (anion-transport protein and protein 4.1). As the result of quantitative changes in these proteins and change in their interrelations in patients with ssential hypertension complicated with metabolic syndrome more intensive disorders of structural and functional organization of red-cell membrane can appear.
format article
author Y. I. Pivovarov
E. E. Kuznetsova
V. G. Gorokhova
A. S. Sergeyeva
I. V. Babushkina
L. B. Koryakina
E. O. Andreyeva
author_facet Y. I. Pivovarov
E. E. Kuznetsova
V. G. Gorokhova
A. S. Sergeyeva
I. V. Babushkina
L. B. Koryakina
E. O. Andreyeva
author_sort Y. I. Pivovarov
title Level of membrane-bound hemoglobin and red-cell membrane proteins in patients with essential hypertension complicated and not complicated with metabolic syndrome
title_short Level of membrane-bound hemoglobin and red-cell membrane proteins in patients with essential hypertension complicated and not complicated with metabolic syndrome
title_full Level of membrane-bound hemoglobin and red-cell membrane proteins in patients with essential hypertension complicated and not complicated with metabolic syndrome
title_fullStr Level of membrane-bound hemoglobin and red-cell membrane proteins in patients with essential hypertension complicated and not complicated with metabolic syndrome
title_full_unstemmed Level of membrane-bound hemoglobin and red-cell membrane proteins in patients with essential hypertension complicated and not complicated with metabolic syndrome
title_sort level of membrane-bound hemoglobin and red-cell membrane proteins in patients with essential hypertension complicated and not complicated with metabolic syndrome
publisher Scientific Сentre for Family Health and Human Reproduction Problems
publishDate 2016
url https://doaj.org/article/aa8ea091c34c41d48206995fadc3dc14
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