MyD88 is a critical regulator of hematopoietic cell-mediated neuroprotection seen after stroke.
Neuroinflammation is critical in the neural cell death seen in stroke. It has been shown that CNS and peripheral responses drive this neuroinflammatory response in the brain. The Toll-like receptors (TLRs) are important regulators of inflammation in response to both exogenous and endogenous stressor...
Guardado en:
Autores principales: | Catherine E Downes, Connie H Y Wong, Katya J Henley, Pedro L Guio-Aguilar, Moses Zhang, Robert Ates, Ashley Mansell, Benjamin T Kile, Peter J Crack |
---|---|
Formato: | article |
Lenguaje: | EN |
Publicado: |
Public Library of Science (PLoS)
2013
|
Materias: | |
Acceso en línea: | https://doaj.org/article/aba7a1673b1c4d669cd0e9194af99a57 |
Etiquetas: |
Agregar Etiqueta
Sin Etiquetas, Sea el primero en etiquetar este registro!
|
Ejemplares similares
-
Identification and function of myeloid differentiation factor 88 (MyD88) in Litopenaeus vannamei.
por: Shuang Zhang, et al.
Publicado: (2012) -
Lipopolysaccharide-induced NF-κB nuclear translocation is primarily dependent on MyD88, but TNFα expression requires TRIF and MyD88
por: Jiro Sakai, et al.
Publicado: (2017) -
MyD88 promotes myoblast fusion in a cell-autonomous manner
por: Sajedah M. Hindi, et al.
Publicado: (2017) -
MyD88 in Macrophages Enhances Liver Fibrosis by Activation of NLRP3 Inflammasome in HSCs
por: Shuang Ge, et al.
Publicado: (2021) -
Basal autophagy prevents autoactivation or enhancement of inflammatory signals by targeting monomeric MyD88
por: Takeshi Into, et al.
Publicado: (2017)