A molecular hypothesis to explain direct and inverse co-morbidities between Alzheimer’s Disease, Glioblastoma and Lung cancer
Abstract Epidemiological studies indicate that patients suffering from Alzheimer’s disease have a lower risk of developing lung cancer, and suggest a higher risk of developing glioblastoma. Here we explore the molecular scenarios that might underlie direct and inverse co-morbidities between these di...
Saved in:
Main Authors: | Jon Sánchez-Valle, Héctor Tejero, Kristina Ibáñez, José Luis Portero, Martin Krallinger, Fátima Al-Shahrour, Rafael Tabarés-Seisdedos, Anaïs Baudot, Alfonso Valencia |
---|---|
Format: | article |
Language: | EN |
Published: |
Nature Portfolio
2017
|
Subjects: | |
Online Access: | https://doaj.org/article/aeb2aaf4301c4094ad5facefc5016e2b |
Tags: |
Add Tag
No Tags, Be the first to tag this record!
|
Similar Items
-
Interpreting molecular similarity between patients as a determinant of disease comorbidity relationships
by: Jon Sánchez-Valle, et al.
Published: (2020) -
The zinc dyshomeostasis hypothesis of Alzheimer's disease.
by: Travis J A Craddock, et al.
Published: (2012) -
Coevolutionary transitions from antagonism to mutualism explained by the Co-Opted Antagonist Hypothesis
by: Christopher A. Johnson, et al.
Published: (2021) -
The hypothesis that Helicobacter pylori predisposes to Alzheimer’s disease is biologically plausible
by: Felice Contaldi, et al.
Published: (2017) -
Hypothesis: Entrapment of lipoprotein particles in the brain causes Alzheimer’s disease
by: Delphine Boche, et al.
Published: (2021)