Productive parvovirus B19 infection of primary human erythroid progenitor cells at hypoxia is regulated by STAT5A and MEK signaling but not HIFα.
Human parvovirus B19 (B19V) causes a variety of human diseases. Disease outcomes of bone marrow failure in patients with high turnover of red blood cells and immunocompromised conditions, and fetal hydrops in pregnant women are resulted from the targeting and destruction of specifically erythroid pr...
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| Autores principales: | , , |
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| Formato: | article |
| Lenguaje: | EN |
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Public Library of Science (PLoS)
2011
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| Acceso en línea: | https://doaj.org/article/b35612a3941c410483b52b7751d8678a |
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