Ccl2/Ccr2 signalling recruits a distinct fetal microchimeric population that rescues delayed maternal wound healing

Foetal microchimeric cells (FMCs) are found in maternal circulation during pregnancy and migrate to injury sites, where they mediate repair, but how this is regulated is unclear. Here, the authors show in mice that the chemokine Ccl2 enhances delayed maternal wound healing through a subpopulation of...

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Autores principales:	Mathieu Castela, Dany Nassar, Maria Sbeih, Marie Jachiet, Zhe Wang, Selim Aractingi
Formato:	article
Lenguaje:	EN
Publicado:	Nature Portfolio 2017
Materias:	Science Q
Acceso en línea:	https://doaj.org/article/b4e128c588f24e50850d686455304093
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spelling	oai:doaj.org-article:b4e128c588f24e50850d6864553040932021-12-02T17:06:17ZCcl2/Ccr2 signalling recruits a distinct fetal microchimeric population that rescues delayed maternal wound healing10.1038/ncomms154632041-1723https://doaj.org/article/b4e128c588f24e50850d6864553040932017-05-01T00:00:00Zhttps://doi.org/10.1038/ncomms15463https://doaj.org/toc/2041-1723Foetal microchimeric cells (FMCs) are found in maternal circulation during pregnancy and migrate to injury sites, where they mediate repair, but how this is regulated is unclear. Here, the authors show in mice that the chemokine Ccl2 enhances delayed maternal wound healing through a subpopulation of Ccr2+ FMCs.Mathieu CastelaDany NassarMaria SbeihMarie JachietZhe WangSelim AractingiNature PortfolioarticleScienceQENNature Communications, Vol 8, Iss 1, Pp 1-13 (2017)
institution	DOAJ
collection	DOAJ
language	EN
topic	Science Q
spellingShingle	Science Q Mathieu Castela Dany Nassar Maria Sbeih Marie Jachiet Zhe Wang Selim Aractingi Ccl2/Ccr2 signalling recruits a distinct fetal microchimeric population that rescues delayed maternal wound healing
description	Foetal microchimeric cells (FMCs) are found in maternal circulation during pregnancy and migrate to injury sites, where they mediate repair, but how this is regulated is unclear. Here, the authors show in mice that the chemokine Ccl2 enhances delayed maternal wound healing through a subpopulation of Ccr2+ FMCs.
format	article
author	Mathieu Castela Dany Nassar Maria Sbeih Marie Jachiet Zhe Wang Selim Aractingi
author_facet	Mathieu Castela Dany Nassar Maria Sbeih Marie Jachiet Zhe Wang Selim Aractingi
author_sort	Mathieu Castela
title	Ccl2/Ccr2 signalling recruits a distinct fetal microchimeric population that rescues delayed maternal wound healing
title_short	Ccl2/Ccr2 signalling recruits a distinct fetal microchimeric population that rescues delayed maternal wound healing
title_full	Ccl2/Ccr2 signalling recruits a distinct fetal microchimeric population that rescues delayed maternal wound healing
title_fullStr	Ccl2/Ccr2 signalling recruits a distinct fetal microchimeric population that rescues delayed maternal wound healing
title_full_unstemmed	Ccl2/Ccr2 signalling recruits a distinct fetal microchimeric population that rescues delayed maternal wound healing
title_sort	ccl2/ccr2 signalling recruits a distinct fetal microchimeric population that rescues delayed maternal wound healing
publisher	Nature Portfolio
publishDate	2017
url	https://doaj.org/article/b4e128c588f24e50850d686455304093
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Ccl2/Ccr2 signalling recruits a distinct fetal microchimeric population that rescues delayed maternal wound healing

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