TMAO: a potential mediator of clopidogrel resistance
Abstract Trimethylamine-N-oxide (TMAO) can activate platelets and increase thrombosis risk in clinical and experimental models. Meanwhile, the patients with coronary artery disease have higher serum TMAO level. However, it remains unknown whether Clopidogrel Resistance (CR) could be attributed to TM...
Guardado en:
Autores principales: | Ruisong Ma, Wenwen Fu, Jing Zhang, Xiaorong Hu, Jun Yang, Hong Jiang |
---|---|
Formato: | article |
Lenguaje: | EN |
Publicado: |
Nature Portfolio
2021
|
Materias: | |
Acceso en línea: | https://doaj.org/article/b6f0fcb212214607899980f1b51ec8cd |
Etiquetas: |
Agregar Etiqueta
Sin Etiquetas, Sea el primero en etiquetar este registro!
|
Ejemplares similares
-
Untargeted metabolomics and transcriptomics identified glutathione metabolism disturbance and PCS and TMAO as potential biomarkers for ER stress in lung
por: Zijing Wang, et al.
Publicado: (2021) -
Resveratrol Attenuates Trimethylamine-<italic toggle="yes">N</italic>-Oxide (TMAO)-Induced Atherosclerosis by Regulating TMAO Synthesis and Bile Acid Metabolism via Remodeling of the Gut Microbiota
por: Ming-liang Chen, et al.
Publicado: (2016) -
Contribution of multidrug and toxin extrusion protein 1 (MATE1) to renal secretion of trimethylamine-N-oxide (TMAO)
por: A. Gessner, et al.
Publicado: (2018) -
Gut microbiota derived trimethylamine N-oxide (TMAO) detection through molecularly imprinted polymer based sensor
por: G. B. V. S. Lakshmi, et al.
Publicado: (2021) -
Risk of postpolypectomy bleeding in patients taking direct oral anticoagulants or clopidogrel
por: Gwang-Un Kim, et al.
Publicado: (2021)