Salicylic acid receptors activate jasmonic acid signalling through a non-canonical pathway to promote effector-triggered immunity

Salicylic acid (SA) and jasmonic acid (JA) often act antagonistically in plant defence. Here, Liu et al. show that during effector-triggered immunity (ETI) against Pseudomonas syringae, JA signalling is activated via a non-canonical pathway involving the SA receptors, NPR3 and NPR4, to positively re...

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Auteurs principaux: Lijing Liu, Fathi-Mohamed Sonbol, Bethany Huot, Yangnan Gu, John Withers, Musoki Mwimba, Jian Yao, Sheng Yang He, Xinnian Dong
Format: article
Langue:EN
Publié: Nature Portfolio 2016
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Accès en ligne:https://doaj.org/article/bccadddfedc74e649b76dd68d6ab8d6c
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Résumé:Salicylic acid (SA) and jasmonic acid (JA) often act antagonistically in plant defence. Here, Liu et al. show that during effector-triggered immunity (ETI) against Pseudomonas syringae, JA signalling is activated via a non-canonical pathway involving the SA receptors, NPR3 and NPR4, to positively regulate ETI.