An innate contribution of human nicotinic receptor polymorphisms to COPD-like lesions

An innate contribution of human nicotinic receptor polymorphisms to COPD-like lesions

Human polymorphisms in nicotinic acetylcholine receptor genes have been linked to both smoking and lung diseases like Chronic Obstructive Pulmonary Disease (COPD) or lung cancer. Here the authors identify a direct role for a human coding polymorphism in COPD-like lesions independent of smoke or nico...

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Autores principales: Julie Routhier, Stéphanie Pons, Mohamed Lamine Freidja, Véronique Dalstein, Jérôme Cutrona, Antoine Jonquet, Nathalie Lalun, Jean-Claude Mérol, Mark Lathrop, Jerry A. Stitzel, Gwenola Kervoaze, Muriel Pichavant, Philippe Gosset, Jean-Marie Tournier, Philippe Birembaut, Valérian Dormoy, Uwe Maskos
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Lenguaje:EN
Publicado: Nature Portfolio 2021
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Acceso en línea:https://doaj.org/article/beb4aa7e77234ba9bf33270113914c9a
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spelling oai:doaj.org-article:beb4aa7e77234ba9bf33270113914c9a2021-11-08T11:06:31ZAn innate contribution of human nicotinic receptor polymorphisms to COPD-like lesions10.1038/s41467-021-26637-62041-1723https://doaj.org/article/beb4aa7e77234ba9bf33270113914c9a2021-11-01T00:00:00Zhttps://doi.org/10.1038/s41467-021-26637-6https://doaj.org/toc/2041-1723Human polymorphisms in nicotinic acetylcholine receptor genes have been linked to both smoking and lung diseases like Chronic Obstructive Pulmonary Disease (COPD) or lung cancer. Here the authors identify a direct role for a human coding polymorphism in COPD-like lesions independent of smoke or nicotine exposure.Julie RouthierStéphanie PonsMohamed Lamine FreidjaVéronique DalsteinJérôme CutronaAntoine JonquetNathalie LalunJean-Claude MérolMark LathropJerry A. StitzelGwenola KervoazeMuriel PichavantPhilippe GossetJean-Marie TournierPhilippe BirembautValérian DormoyUwe MaskosNature PortfolioarticleScienceQENNature Communications, Vol 12, Iss 1, Pp 1-13 (2021)
institution DOAJ
collection DOAJ
language EN
topic Science
Q
spellingShingle Science
Q
Julie Routhier
Stéphanie Pons
Mohamed Lamine Freidja
Véronique Dalstein
Jérôme Cutrona
Antoine Jonquet
Nathalie Lalun
Jean-Claude Mérol
Mark Lathrop
Jerry A. Stitzel
Gwenola Kervoaze
Muriel Pichavant
Philippe Gosset
Jean-Marie Tournier
Philippe Birembaut
Valérian Dormoy
Uwe Maskos
An innate contribution of human nicotinic receptor polymorphisms to COPD-like lesions
description Human polymorphisms in nicotinic acetylcholine receptor genes have been linked to both smoking and lung diseases like Chronic Obstructive Pulmonary Disease (COPD) or lung cancer. Here the authors identify a direct role for a human coding polymorphism in COPD-like lesions independent of smoke or nicotine exposure.
format article
author Julie Routhier
Stéphanie Pons
Mohamed Lamine Freidja
Véronique Dalstein
Jérôme Cutrona
Antoine Jonquet
Nathalie Lalun
Jean-Claude Mérol
Mark Lathrop
Jerry A. Stitzel
Gwenola Kervoaze
Muriel Pichavant
Philippe Gosset
Jean-Marie Tournier
Philippe Birembaut
Valérian Dormoy
Uwe Maskos
author_facet Julie Routhier
Stéphanie Pons
Mohamed Lamine Freidja
Véronique Dalstein
Jérôme Cutrona
Antoine Jonquet
Nathalie Lalun
Jean-Claude Mérol
Mark Lathrop
Jerry A. Stitzel
Gwenola Kervoaze
Muriel Pichavant
Philippe Gosset
Jean-Marie Tournier
Philippe Birembaut
Valérian Dormoy
Uwe Maskos
author_sort Julie Routhier
title An innate contribution of human nicotinic receptor polymorphisms to COPD-like lesions
title_short An innate contribution of human nicotinic receptor polymorphisms to COPD-like lesions
title_full An innate contribution of human nicotinic receptor polymorphisms to COPD-like lesions
title_fullStr An innate contribution of human nicotinic receptor polymorphisms to COPD-like lesions
title_full_unstemmed An innate contribution of human nicotinic receptor polymorphisms to COPD-like lesions
title_sort innate contribution of human nicotinic receptor polymorphisms to copd-like lesions
publisher Nature Portfolio
publishDate 2021
url https://doaj.org/article/beb4aa7e77234ba9bf33270113914c9a
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