Integrated TORC1 and PKA signaling control the temporal activation of glucose-induced gene expression in yeast
Yeast cells respond to nutrients by altering expression of the protein synthesis genes and thus their growth rate. Here, the authors use microarrays to show that TORC1 controls gene expression during steady state growth while PKA speeds up expression changes when nutrient levels change.
Saved in:
Main Authors: | Joseph Kunkel, Xiangxia Luo, Andrew P. Capaldi |
---|---|
Format: | article |
Language: | EN |
Published: |
Nature Portfolio
2019
|
Subjects: | |
Online Access: | https://doaj.org/article/c2f0f6643f4a4866a44a494c9713d2d7 |
Tags: |
Add Tag
No Tags, Be the first to tag this record!
|
Similar Items
-
A novel connection between the Cell Wall Integrity and the PKA pathways regulates cell wall stress response in yeast
by: Raúl García, et al.
Published: (2017) -
Bimodal antagonism of PKA signalling by ARHGAP36
by: Rebecca L. Eccles, et al.
Published: (2016) -
The GCN5-CITED2-PKA signalling module controls hepatic glucose metabolism through a cAMP-induced substrate switch
by: Mashito Sakai, et al.
Published: (2016) -
Integration of global signaling pathways, cAMP-PKA, MAPK and TOR in the regulation of FLO11.
by: P K Vinod, et al.
Published: (2008) -
Differential controls of MAIT cell effector polarization by mTORC1/mTORC2 via integrating cytokine and costimulatory signals
by: Huishan Tao, et al.
Published: (2021)