Determining the effect of aging, recovery time, and post-stroke memantine treatment on delayed thalamic gliosis after cortical infarct
Abstract Secondary injury following cortical stroke includes delayed gliosis and eventual neuronal loss in the thalamus. However, the effects of aging and the potential to ameliorate this gliosis with NMDA receptor (NMDAR) antagonism are not established. We used the permanent distal middle cerebral...
Guardado en:
Autores principales: | Gab Seok Kim, Jessica M. Stephenson, Abdullah Al Mamun, Ting Wu, Monica G. Goss, Jia-Wei Min, Jun Li, Fudong Liu, Sean P. Marrelli |
---|---|
Formato: | article |
Lenguaje: | EN |
Publicado: |
Nature Portfolio
2021
|
Materias: | |
Acceso en línea: | https://doaj.org/article/d532eb5950704af0b83e9a992a4a9c7a |
Etiquetas: |
Agregar Etiqueta
Sin Etiquetas, Sea el primero en etiquetar este registro!
|
Ejemplares similares
-
Stroke subtype-dependent synapse elimination by reactive gliosis in mice
por: Xiaojing Shi, et al.
Publicado: (2021) -
Thalamic Bursts Down-regulate Cortical Theta and Nociceptive Behavior
por: Brian W. LeBlanc, et al.
Publicado: (2017) -
Sensorimotor stroke alters hippocampo-thalamic network activity
por: Philipp Baumgartner, et al.
Publicado: (2018) -
Clinical Analysis of Bilateral Thalamic Infarction Caused by Percheron Artery Occlusion
por: Chen P, et al.
Publicado: (2021) -
Regulation of cortical activity and arousal by the matrix cells of the ventromedial thalamic nucleus
por: Sakiko Honjoh, et al.
Publicado: (2018)