GPR55 deletion in mice leads to age-related ventricular dysfunction and impaired adrenoceptor-mediated inotropic responses.
G protein coupled receptor 55 (GPR55) is expressed throughout the body, and although its exact physiological function is unknown, studies have suggested a role in the cardiovascular system. In particular, GPR55 has been proposed as mediating the haemodynamic effects of a number of atypical cannabino...
Enregistré dans:
Auteurs principaux: | Sarah K Walsh, Emma E Hector, Anne-Christine Andréasson, Ann-Cathrine Jönsson-Rylander, Cherry L Wainwright |
---|---|
Format: | article |
Langue: | EN |
Publié: |
Public Library of Science (PLoS)
2014
|
Sujets: | |
Accès en ligne: | https://doaj.org/article/dd26c54342f04552a5b6c5045d338fcb |
Tags: |
Ajouter un tag
Pas de tags, Soyez le premier à ajouter un tag!
|
Documents similaires
-
Inflammatory and Neuropathic Nociception is Preserved in GPR55 Knockout Mice
par: Lawrence M. Carey, et autres
Publié: (2017) -
Haematopoietic and cardiac GPR55 synchronize post-myocardial infarction remodelling
par: Sarah-Lena Puhl, et autres
Publié: (2021) -
Targeting Oxidative Stress: Novel Coumarin-Based Inverse Agonists of GPR55
par: Matthias Apweiler, et autres
Publié: (2021) -
Caveolin contributes to the modulation of basal and β-adrenoceptor stimulated function of the adult rat ventricular myocyte by simvastatin: a novel pleiotropic effect.
par: Sara D Pugh, et autres
Publié: (2014) -
Cerebral effects of commonly used vasopressor-inotropes: a study in newborn piglets.
par: Gitte H Hahn, et autres
Publié: (2013)