Hepatic Knockdown of Endothelin Type A Receptor (ETAR) Ameliorates Hepatic Insulin Resistance and Hyperglycemia Through Suppressing p66Shc-Mediated Mitochondrial Fragmentation in High-Fat Diet-Fed Mice

Li Feng,* Songhua Wang,* Feng Chen,* Cheng Zhang, Qiao Wang, Yuting Zhao, Zifeng Zhang School of Life Science, Jiangsu Normal University, Xuzhou, Jiangsu, 221116, People’s Republic of China*These authors contributed equally to this workCorrespondence: Zifeng Zhang 101 Shanghai Road, Xuzhou...

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Bibliographic Details
Main Authors:	Feng L, Wang S, Chen F, Zhang C, Wang Q, Zhao Y, Zhang Z
Format:	article
Language:	EN
Published:	Dove Medical Press 2021
Subjects:	endothelin-1 endothelin type a receptor 66-kda src homology 2 domain-containing protein mitochondrial fragmentation metabolism abnormality Specialties of internal medicine RC581-951
Online Access:	https://doaj.org/article/e92b0bfd164c47ca8efa62a478f61a8c
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Hepatic Knockdown of Endothelin Type A Receptor (ETAR) Ameliorates Hepatic Insulin Resistance and Hyperglycemia Through Suppressing p66Shc-Mediated Mitochondrial Fragmentation in High-Fat Diet-Fed Mice

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