Glucocorticoids protect HEI-OC1 cells from tunicamycin-induced cell damage via inhibiting endoplasmic reticulum stress

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Glucocorticoids protect HEI-OC1 cells from tunicamycin-induced cell damage via inhibiting endoplasmic reticulum stress

To analyze mechanisms of action of glucocorticoid treatment for endoplasmic reticulum stress (ERS) in sensorineural hearing loss (SNHL), we aimed to evaluate the expression and activation status of the protein kinase RNA-like ER kinase (PERK)–C/EBP homologous protein (CHOP) pathway, which is the maj...

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Auteurs principaux:	Liu Zhibiao, Fei Bing, Xie Lisheng, Liu Jin, Chen Xiaorui, Zhu Wenyan, Lv Lingyun, Ma Wei, Gao Ziwen, Hou Jie, She Wandong
Format:	article
Langue:	EN
Publié:	De Gruyter 2021
Sujets:	endoplasmic reticulum stress perk–chop pathway dexamethasone mifepristone Biology (General) QH301-705.5
Accès en ligne:	https://doaj.org/article/eba06e7f7ce149bc9839bf9fc165fc95
Tags:	Ajouter un tag Pas de tags, Soyez le premier à ajouter un tag!

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