Allicin disrupts cardiac Cav1.2 channels via trafficking

Context: Allicin is a potential antiarrhythmic agent. The antiarrhythmic properties of allicin rely on its blockade of various cardiac ion channels. The l-type calcium (Cav1.2) channel provides a pivotal substrate for cardiac electrophysiologic activities. The mechanism of allicin on Cav1.2 remains...

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Autores principales: Dan Han, Lingping Xu, Peng Liu, Yingying Liu, Chaofeng Sun, Yanrong Yin
Formato: article
Lenguaje:EN
Publicado: Taylor & Francis Group 2019
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Acceso en línea:https://doaj.org/article/f3bad6817d204abb9efb23e6c54424af
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