Potencia de los músculos inspiratorios en insuficiencia cardíaca crónica y en enfermedad pulmonar obstructiva crónica

Background: The maximal pressure generated by inspiratory muscles (PIMax) is an index of their strength which is diminished in both chronic obstructive pulmonary disease (COPD) and chronic heart failure (CHF). Although inspiratory muscle power output (IMPO), which includes both strength and velocity...

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Autores principales:	Mangelsdorff G,Günther, Borzone T,Gisella, Leiva G,Alicia, Martínez S,Alejandro, Lisboa B,Carmen
Lenguaje:	Spanish / Castilian
Publicado:	Sociedad Médica de Santiago 2001
Materias:	Heart failure, congestive Lung diseases, obstructive Respiratory function tests Respiratory mechanism Respiratory muscles
Acceso en línea:	http://www.scielo.cl/scielo.php?script=sci_arttext&pid=S0034-98872001000100007
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spelling	oai:scielo:S0034-988720010001000072005-11-23Potencia de los músculos inspiratorios en insuficiencia cardíaca crónica y en enfermedad pulmonar obstructiva crónicaMangelsdorff G,GüntherBorzone T,GisellaLeiva G,AliciaMartínez S,AlejandroLisboa B,Carmen Heart failure, congestive Lung diseases, obstructive Respiratory function tests Respiratory mechanism Respiratory muscles Background: The maximal pressure generated by inspiratory muscles (PIMax) is an index of their strength which is diminished in both chronic obstructive pulmonary disease (COPD) and chronic heart failure (CHF). Although inspiratory muscle power output (IMPO), which includes both strength and velocity of shortening, has been shown to be reduced in COPD, there is no information regarding IMPO in CHF. Aim: To measure Impo in patients with CHF and COPD. Patients and methods: We studied 9 CHF patients with functional capacity II and III and 9 patients with severe COPD. Eight normal subjects of similar ages were included as controls. Power output was measured using the incremental threshold loading test. Results: Maximal IMPO was significantly reduced in both groups of patients. Power output developed with each increasing load was also diminished, basically as a consequence of a reduction in <IMG SRC="../img/v.gif" WIDTH=9 HEIGHT=12>insp. The degree of dyspnea at the end of the test was greater in COPD than in CHF patients and normal subjects. For a given level of power, dyspnea was also greater in patients than in normals subjects. There was no decrease in SpO2 during the test. Conclusions: IMPO is equally reduced in COPD and CHF patients. Power output is better related to dyspnea than PIMax, probably because of the inclusion of shortening velocity (Rev Méd Chile 2001; 129: 51-59).info:eu-repo/semantics/openAccessSociedad Médica de SantiagoRevista médica de Chile v.129 n.1 20012001-01-01text/htmlhttp://www.scielo.cl/scielo.php?script=sci_arttext&pid=S0034-98872001000100007es10.4067/S0034-98872001000100007
institution	Scielo Chile
collection	Scielo Chile
language	Spanish / Castilian
topic	Heart failure, congestive Lung diseases, obstructive Respiratory function tests Respiratory mechanism Respiratory muscles
spellingShingle	Heart failure, congestive Lung diseases, obstructive Respiratory function tests Respiratory mechanism Respiratory muscles Mangelsdorff G,Günther Borzone T,Gisella Leiva G,Alicia Martínez S,Alejandro Lisboa B,Carmen Potencia de los músculos inspiratorios en insuficiencia cardíaca crónica y en enfermedad pulmonar obstructiva crónica
description	Background: The maximal pressure generated by inspiratory muscles (PIMax) is an index of their strength which is diminished in both chronic obstructive pulmonary disease (COPD) and chronic heart failure (CHF). Although inspiratory muscle power output (IMPO), which includes both strength and velocity of shortening, has been shown to be reduced in COPD, there is no information regarding IMPO in CHF. Aim: To measure Impo in patients with CHF and COPD. Patients and methods: We studied 9 CHF patients with functional capacity II and III and 9 patients with severe COPD. Eight normal subjects of similar ages were included as controls. Power output was measured using the incremental threshold loading test. Results: Maximal IMPO was significantly reduced in both groups of patients. Power output developed with each increasing load was also diminished, basically as a consequence of a reduction in <IMG SRC="../img/v.gif" WIDTH=9 HEIGHT=12>insp. The degree of dyspnea at the end of the test was greater in COPD than in CHF patients and normal subjects. For a given level of power, dyspnea was also greater in patients than in normals subjects. There was no decrease in SpO2 during the test. Conclusions: IMPO is equally reduced in COPD and CHF patients. Power output is better related to dyspnea than PIMax, probably because of the inclusion of shortening velocity (Rev Méd Chile 2001; 129: 51-59).
author	Mangelsdorff G,Günther Borzone T,Gisella Leiva G,Alicia Martínez S,Alejandro Lisboa B,Carmen
author_facet	Mangelsdorff G,Günther Borzone T,Gisella Leiva G,Alicia Martínez S,Alejandro Lisboa B,Carmen
author_sort	Mangelsdorff G,Günther
title	Potencia de los músculos inspiratorios en insuficiencia cardíaca crónica y en enfermedad pulmonar obstructiva crónica
title_short	Potencia de los músculos inspiratorios en insuficiencia cardíaca crónica y en enfermedad pulmonar obstructiva crónica
title_full	Potencia de los músculos inspiratorios en insuficiencia cardíaca crónica y en enfermedad pulmonar obstructiva crónica
title_fullStr	Potencia de los músculos inspiratorios en insuficiencia cardíaca crónica y en enfermedad pulmonar obstructiva crónica
title_full_unstemmed	Potencia de los músculos inspiratorios en insuficiencia cardíaca crónica y en enfermedad pulmonar obstructiva crónica
title_sort	potencia de los músculos inspiratorios en insuficiencia cardíaca crónica y en enfermedad pulmonar obstructiva crónica
publisher	Sociedad Médica de Santiago
publishDate	2001
url	http://www.scielo.cl/scielo.php?script=sci_arttext&pid=S0034-98872001000100007
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Potencia de los músculos inspiratorios en insuficiencia cardíaca crónica y en enfermedad pulmonar obstructiva crónica

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