Función de los músculos inspiratorios, parámetros hemodinámicos y disnea en pacientes con estenosis mitral

Background: The mechanism involved in dyspnea in patients with mitral valve stenosis (MS) is not completely understood. Aim: To evaluate in patients with MS, changes in hemodynamic parameters during the assessment of inspiratory muscle endurance (IME) and the relationship between IME, hemodynamics a...

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Autores principales:	Palacios M,Sylvia, Pérez P,Osvaldo, Pino R,Jorge, Carrasco,Gonzalo, Fasce,Eduardo, Olmos C,Alfonso
Lenguaje:	Spanish / Castilian
Publicado:	Sociedad Médica de Santiago 2006
Materias:	Hemodynamic phenomena Mitral valve stenosis Respiratory function tests
Acceso en línea:	http://www.scielo.cl/scielo.php?script=sci_arttext&pid=S0034-98872006000500003
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spelling	oai:scielo:S0034-988720060005000032006-06-22Función de los músculos inspiratorios, parámetros hemodinámicos y disnea en pacientes con estenosis mitralPalacios M,SylviaPérez P,OsvaldoPino R,JorgeCarrasco,GonzaloFasce,EduardoOlmos C,Alfonso Hemodynamic phenomena Mitral valve stenosis Respiratory function tests Background: The mechanism involved in dyspnea in patients with mitral valve stenosis (MS) is not completely understood. Aim: To evaluate in patients with MS, changes in hemodynamic parameters during the assessment of inspiratory muscle endurance (IME) and the relationship between IME, hemodynamics and dyspnea. Subjects and methods: We studied 13 patients (9 in NYHA class II and 4 in class III). Endurance was evaluated using a two minute incremental threshold loading test, to obtain the maximal sustainable inspiratory pressure (SIP), and maximal inspiratory load (MIL). During the test, cardiac output (CO), mean pulmonary and capillary pressures (PAP and PCP, respectively), were evaluated. Results: Compared to six normal subjects, MS patients had reduced SIP and MIL (p <0.01), which correlated with baseline index of dyspnea (r= 0.57 and r=0.52, respectively, p <0.05). At the end of the test period, basal CO, cardiac index (CI), PAP and PCP increased from 3.4 to 4.0 l/min-1; 2.1 to 2.5 l/min-1/m-2; 15 to 25 and 11 to 18 mmHg, respectively (p <0.01). No relationship between IME and hemodynamic parameters was found. Conclusions: IME is reduced and is closely related to dyspnea in these patients with MS. The observed low CI, suggests that muscle underperfusion could contribute to this dysfunction during the inspiratoryinfo:eu-repo/semantics/openAccessSociedad Médica de SantiagoRevista médica de Chile v.134 n.5 20062006-05-01text/htmlhttp://www.scielo.cl/scielo.php?script=sci_arttext&pid=S0034-98872006000500003es10.4067/S0034-98872006000500003
institution	Scielo Chile
collection	Scielo Chile
language	Spanish / Castilian
topic	Hemodynamic phenomena Mitral valve stenosis Respiratory function tests
spellingShingle	Hemodynamic phenomena Mitral valve stenosis Respiratory function tests Palacios M,Sylvia Pérez P,Osvaldo Pino R,Jorge Carrasco,Gonzalo Fasce,Eduardo Olmos C,Alfonso Función de los músculos inspiratorios, parámetros hemodinámicos y disnea en pacientes con estenosis mitral
description	Background: The mechanism involved in dyspnea in patients with mitral valve stenosis (MS) is not completely understood. Aim: To evaluate in patients with MS, changes in hemodynamic parameters during the assessment of inspiratory muscle endurance (IME) and the relationship between IME, hemodynamics and dyspnea. Subjects and methods: We studied 13 patients (9 in NYHA class II and 4 in class III). Endurance was evaluated using a two minute incremental threshold loading test, to obtain the maximal sustainable inspiratory pressure (SIP), and maximal inspiratory load (MIL). During the test, cardiac output (CO), mean pulmonary and capillary pressures (PAP and PCP, respectively), were evaluated. Results: Compared to six normal subjects, MS patients had reduced SIP and MIL (p <0.01), which correlated with baseline index of dyspnea (r= 0.57 and r=0.52, respectively, p <0.05). At the end of the test period, basal CO, cardiac index (CI), PAP and PCP increased from 3.4 to 4.0 l/min-1; 2.1 to 2.5 l/min-1/m-2; 15 to 25 and 11 to 18 mmHg, respectively (p <0.01). No relationship between IME and hemodynamic parameters was found. Conclusions: IME is reduced and is closely related to dyspnea in these patients with MS. The observed low CI, suggests that muscle underperfusion could contribute to this dysfunction during the inspiratory
author	Palacios M,Sylvia Pérez P,Osvaldo Pino R,Jorge Carrasco,Gonzalo Fasce,Eduardo Olmos C,Alfonso
author_facet	Palacios M,Sylvia Pérez P,Osvaldo Pino R,Jorge Carrasco,Gonzalo Fasce,Eduardo Olmos C,Alfonso
author_sort	Palacios M,Sylvia
title	Función de los músculos inspiratorios, parámetros hemodinámicos y disnea en pacientes con estenosis mitral
title_short	Función de los músculos inspiratorios, parámetros hemodinámicos y disnea en pacientes con estenosis mitral
title_full	Función de los músculos inspiratorios, parámetros hemodinámicos y disnea en pacientes con estenosis mitral
title_fullStr	Función de los músculos inspiratorios, parámetros hemodinámicos y disnea en pacientes con estenosis mitral
title_full_unstemmed	Función de los músculos inspiratorios, parámetros hemodinámicos y disnea en pacientes con estenosis mitral
title_sort	función de los músculos inspiratorios, parámetros hemodinámicos y disnea en pacientes con estenosis mitral
publisher	Sociedad Médica de Santiago
publishDate	2006
url	http://www.scielo.cl/scielo.php?script=sci_arttext&pid=S0034-98872006000500003
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Función de los músculos inspiratorios, parámetros hemodinámicos y disnea en pacientes con estenosis mitral

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