Encefalopatía posterior reversible como primera manifestación del Síndrome de Guillain-Barré

Background: We report a 56year old male hypertensive, who presented with a posterior reversible encephalopathy syndrome (PRES) as an initial manifestation of Guillain-Barré syndrome (GBS). His first symptoms were right hemiparesis and hemihypoesthesia, followed by headache, dizziness, dysarthria and...

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Autores principales:	Urrutia L,Sergio, Venegas R,Eduardo, Figueroa V,Cristián, Carrizo C,Catalina
Lenguaje:	Spanish / Castilian
Publicado:	Sociedad Médica de Santiago 2012
Materias:	Guillain-Barré syndrome Hypertensive encephalopathy Posterior leukoencephalopathy syndrome
Acceso en línea:	http://www.scielo.cl/scielo.php?script=sci_arttext&pid=S0034-98872012001000012
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spelling	oai:scielo:S0034-988720120010000122013-03-18Encefalopatía posterior reversible como primera manifestación del Síndrome de Guillain-BarréUrrutia L,SergioVenegas R,EduardoFigueroa V,CristiánCarrizo C,Catalina Guillain-Barré syndrome Hypertensive encephalopathy Posterior leukoencephalopathy syndrome Background: We report a 56year old male hypertensive, who presented with a posterior reversible encephalopathy syndrome (PRES) as an initial manifestation of Guillain-Barré syndrome (GBS). His first symptoms were right hemiparesis and hemihypoesthesia, followed by headache, dizziness, dysarthria and a general feeling of discomfort. On the third day, flaccid tetraparesis, impairment of consciousness, epileptic seizures and respiratory failure appeared, along with severe hypertension. Cerebral Magnetic Resonance Imaging showed the characteristic PRES lesions. Cerebrospinal fluid analyses revealed albumin-cytological dissociation and nerve conduction studies showed an axonal demyelinating polyradiculoneuropathy, which confirmed the diagnosis of GBS. Treatment with intravenous immunoglobulin was given together with antihypertensive therapy and mechanical ventilation, achieving an important clinical and imaging remission of PRES, but maintaining tetraparesis during the hospitalization. Twelve months after discharge and regular motor rehabilitation, the patient achieved complete autonomy on the activities of daily living. It has been postulated that the autonomic failure and the elevation of circulating pro-inflammatory cytokines in GBS may be the cause of a breach in the blood-brain barrier, thus causing PRES, that can completely remit with an adequate management.info:eu-repo/semantics/openAccessSociedad Médica de SantiagoRevista médica de Chile v.140 n.10 20122012-10-01text/htmlhttp://www.scielo.cl/scielo.php?script=sci_arttext&pid=S0034-98872012001000012es10.4067/S0034-98872012001000012
institution	Scielo Chile
collection	Scielo Chile
language	Spanish / Castilian
topic	Guillain-Barré syndrome Hypertensive encephalopathy Posterior leukoencephalopathy syndrome
spellingShingle	Guillain-Barré syndrome Hypertensive encephalopathy Posterior leukoencephalopathy syndrome Urrutia L,Sergio Venegas R,Eduardo Figueroa V,Cristián Carrizo C,Catalina Encefalopatía posterior reversible como primera manifestación del Síndrome de Guillain-Barré
description	Background: We report a 56year old male hypertensive, who presented with a posterior reversible encephalopathy syndrome (PRES) as an initial manifestation of Guillain-Barré syndrome (GBS). His first symptoms were right hemiparesis and hemihypoesthesia, followed by headache, dizziness, dysarthria and a general feeling of discomfort. On the third day, flaccid tetraparesis, impairment of consciousness, epileptic seizures and respiratory failure appeared, along with severe hypertension. Cerebral Magnetic Resonance Imaging showed the characteristic PRES lesions. Cerebrospinal fluid analyses revealed albumin-cytological dissociation and nerve conduction studies showed an axonal demyelinating polyradiculoneuropathy, which confirmed the diagnosis of GBS. Treatment with intravenous immunoglobulin was given together with antihypertensive therapy and mechanical ventilation, achieving an important clinical and imaging remission of PRES, but maintaining tetraparesis during the hospitalization. Twelve months after discharge and regular motor rehabilitation, the patient achieved complete autonomy on the activities of daily living. It has been postulated that the autonomic failure and the elevation of circulating pro-inflammatory cytokines in GBS may be the cause of a breach in the blood-brain barrier, thus causing PRES, that can completely remit with an adequate management.
author	Urrutia L,Sergio Venegas R,Eduardo Figueroa V,Cristián Carrizo C,Catalina
author_facet	Urrutia L,Sergio Venegas R,Eduardo Figueroa V,Cristián Carrizo C,Catalina
author_sort	Urrutia L,Sergio
title	Encefalopatía posterior reversible como primera manifestación del Síndrome de Guillain-Barré
title_short	Encefalopatía posterior reversible como primera manifestación del Síndrome de Guillain-Barré
title_full	Encefalopatía posterior reversible como primera manifestación del Síndrome de Guillain-Barré
title_fullStr	Encefalopatía posterior reversible como primera manifestación del Síndrome de Guillain-Barré
title_full_unstemmed	Encefalopatía posterior reversible como primera manifestación del Síndrome de Guillain-Barré
title_sort	encefalopatía posterior reversible como primera manifestación del síndrome de guillain-barré
publisher	Sociedad Médica de Santiago
publishDate	2012
url	http://www.scielo.cl/scielo.php?script=sci_arttext&pid=S0034-98872012001000012
work_keys_str_mv	AT urrutialsergio encefalopatiaposteriorreversiblecomoprimeramanifestaciondelsindromedeguillainbarre AT venegasreduardo encefalopatiaposteriorreversiblecomoprimeramanifestaciondelsindromedeguillainbarre AT figueroavcristian encefalopatiaposteriorreversiblecomoprimeramanifestaciondelsindromedeguillainbarre AT carrizoccatalina encefalopatiaposteriorreversiblecomoprimeramanifestaciondelsindromedeguillainbarre
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Encefalopatía posterior reversible como primera manifestación del Síndrome de Guillain-Barré

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