Excess circulating alternatively activated myeloid (M2) cells accelerate ALS progression while inhibiting experimental autoimmune encephalomyelitis.

por Ilan Vaknin, Gilad Kunis, Omer Miller, Oleg Butovsky, Shay Bukshpan, David R Beers, Jenny S Henkel, Eti Yoles, Stanley H Appel, Michal Schwartz
Publicado 2011