Kerstin Lindblad-Toh

Lindblad-Toh in 2013 Kerstin Lindblad-Toh is a scientist in comparative genomics, specializing in mammalian genetics. She is the Scientific Director of vertebrate genomics at the Broad Institute and a professor in comparative genomics at Uppsala University. In 2010 she co-founded Science for Life Laboratory (SciLifeLab) together with Mathias Uhlén and acted as Co-Director until 2015. As the leader of the Broad Institute's Mammalian Genome Initiative she has led the effort to sequence and analyze the genomes of various mammals, including mouse, dog, chimpanzee, horse, rabbit and opossum. She has researched extensively on the genetics of dogs, identifying genes and genetic variants important in disease susceptibility, morphology and behavior.

In 2015, she started the Zoonomia Project, named after the medical work (1794–96) by Erasmus Darwin.

Lindblad-Toh is elected to the National Academy of Sciences of the United States and the Royal Swedish Academy of Sciences. Provided by Wikipedia
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  1. 1
    Identification and classification of conserved RNA secondary structures in the human genome.
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    A frameshift mutation in golden retriever dogs with progressive retinal atrophy endorses SLC4A3 as a candidate gene for human retinal degenerations.
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    A deletion in the N-myc downstream regulated gene 1 (NDRG1) gene in Greyhounds with polyneuropathy.
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    The ABCC4 gene is associated with pyometra in golden retriever dogs
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    Author Correction: Targeted sequencing reveals the somatic mutation landscape in a Swedish breast cancer cohort
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    A simple repeat polymorphism in the MITF-M promoter is a key regulator of white spotting in dogs.
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    A novel canine reference genome resolves genomic architecture and uncovers transcript complexity
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    Thorough investigation of a canine autoinflammatory disease (AID) confirms one main risk locus and suggests a modifier locus for amyloidosis.
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    The genetic consequences of dog breed formation-Accumulation of deleterious genetic variation and fixation of mutations associated with myxomatous mitral valve disease in cavalier King Charles spaniels.
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    ZBED6, a novel transcription factor derived from a domesticated DNA transposon regulates IGF2 expression and muscle growth.
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    Lineage-specific biology revealed by a finished genome assembly of the mouse.
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    Integrating evolutionary and regulatory information with a multispecies approach implicates genes and pathways in obsessive-compulsive disorder
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