Random amino acid mutations and protein misfolding lead to Shannon limit in sequence-structure communication.
The transmission of genomic information from coding sequence to protein structure during protein synthesis is subject to stochastic errors. To analyze transmission limits in the presence of spurious errors, Shannon's noisy channel theorem is applied to a communication channel between amino acid...
Guardado en:
Autor principal: | Andreas Martin Lisewski |
---|---|
Formato: | article |
Lenguaje: | EN |
Publicado: |
Public Library of Science (PLoS)
2008
|
Materias: | |
Acceso en línea: | https://doaj.org/article/22e56c9f285348d6b6f2b56b1322e013 |
Etiquetas: |
Agregar Etiqueta
Sin Etiquetas, Sea el primero en etiquetar este registro!
|
Ejemplares similares
-
The non-protein amino acid BMAA is misincorporated into human proteins in place of L-serine causing protein misfolding and aggregation.
por: Rachael Anne Dunlop, et al.
Publicado: (2013) -
Recurrent background mutations in WHI2 impair proteostasis and degradation of misfolded cytosolic proteins in Saccharomyces cerevisiae
por: Sophie A. Comyn, et al.
Publicado: (2017) -
Taxonomy of Mitochondrial Cytochrome B Proteins of the Same Amino Acid Sequence Length
por: Alexander A. Zamyatnin, et al.
Publicado: (2021) -
Structure network-based landscape of rhodopsin misfolding by mutations and algorithmic prediction of small chaperone action
por: Angelo Felline, et al.
Publicado: (2021) -
The Cytotoxicity and Clearance of Mutant Huntingtin and Other Misfolded Proteins
por: Austin Folger, et al.
Publicado: (2021)