Expanded Hemodialysis Therapy Ameliorates Uremia-Induced Systemic Microinflammation and Endothelial Dysfunction by Modulating VEGF, TNF-α and AP-1 Signaling

Expanded Hemodialysis Therapy Ameliorates Uremia-Induced Systemic Microinflammation and Endothelial Dysfunction by Modulating VEGF, TNF-α and AP-1 Signaling

AbstractSystemic chronic microinflammation and altered cytokine signaling, with adjunct cardiovascular disease (CVD), endothelial maladaptation and dysfunction is common in dialysis patients suffering from end-stage renal disease and associated with increased morbidity and mortality. New hemodialysi...

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Détails bibliographiques
Auteurs principaux: Rusan Catar, Guido Moll, Julian Kamhieh-Milz, Christian Luecht, Lei Chen, Hongfan Zhao, Lucas Ernst, Kevin Willy, Matthias Girndt, Roman Fiedler, Janusz Witowski, Henning Morawietz, Olle Ringdén, Duska Dragun, Kai-Uwe Eckardt, Ralf Schindler, Daniel Zickler
Format: article
Langue:EN
Publié: Frontiers Media S.A. 2021
Sujets:
cardiovascular disease
endothelial cell (dys)function
expanded hemodialysis therapy
chronic kidney disease
end-stage renal disease
uremic toxins / systemic microinflammation
Immunologic diseases. Allergy
RC581-607
Accès en ligne:https://doaj.org/article/2b5da9f8659e4b1c889dc9aee829ea9d
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https://doaj.org/article/2b5da9f8659e4b1c889dc9aee829ea9d

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