Keratinocyte-targeted overexpression of the glucocorticoid receptor delays cutaneous wound healing.

Delayed wound healing is one of the most common secondary adverse effects associated to the therapeutic use of glucocorticoid (GC) analogs, which act through the ligand-dependent transcription factor GC-receptor (GR). GR function is exerted through DNA-binding-dependent and -independent mechanisms,...

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Autores principales: Ana Sanchis, Lorena Alba, Víctor Latorre, Lisa M Sevilla, Paloma Pérez
Formato: article
Lenguaje:EN
Publicado: Public Library of Science (PLoS) 2012
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Acceso en línea:https://doaj.org/article/3b0ab5a17fa540bea7d6d57ece77275a
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