Platelet Phenotype Analysis of COVID-19 Patients Reveals Progressive Changes in the Activation of Integrin αIIbβ3, F13A1, the SARS-CoV-2 Target EIF4A1 and Annexin A5

Background: The fatal consequences of an infection with severe acute respiratory syndrome coronavirus 2 are not only caused by severe pneumonia, but also by thrombosis. Platelets are important regulators of thrombosis, but their involvement in the pathogenesis of COVID-19 is largely unknown. The aim...

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Autores principales:	Huriye Ercan, Waltraud Cornelia Schrottmaier, Anita Pirabe, Anna Schmuckenschlager, David Pereyra, Jonas Santol, Erich Pawelka, Marianna T. Traugott, Christian Schörgenhofer, Tamara Seitz, Mario Karolyi, Jae-Won Yang, Bernd Jilma, Alexander Zoufaly, Alice Assinger, Maria Zellner
Formato:	article
Lenguaje:	EN
Publicado:	Frontiers Media S.A. 2021
Materias:	COVID-19 thrombosis platelets integrin αIIbβ3 coagulation factor XIII (FXIII, F13A1) antiphospholipid syndrome Diseases of the circulatory (Cardiovascular) system RC666-701
Acceso en línea:	https://doaj.org/article/3b745cc794fd43db9f5982030c9086d2
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Platelet Phenotype Analysis of COVID-19 Patients Reveals Progressive Changes in the Activation of Integrin αIIbβ3, F13A1, the SARS-CoV-2 Target EIF4A1 and Annexin A5

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