Developmental exposure of mice to dioxin promotes transgenerational testicular inflammation and an increased risk of preterm birth in unexposed mating partners.
TCDD (2,3,7,8-tetrachlorodibenzo-p-dioxin, commonly known as dioxin) is a ubiquitous environmental contaminant and known endocrine disruptor. Using a mouse model, we previously found that adult female mice exposed in utero to TCDD (F1 generation) as well as multiple subsequent generations (F2-F4) ex...
Guardado en:
Autores principales: | Kaylon L Bruner-Tran, Tianbing Ding, Kallie B Yeoman, Anthony Archibong, Joe A Arosh, Kevin G Osteen |
---|---|
Formato: | article |
Lenguaje: | EN |
Publicado: |
Public Library of Science (PLoS)
2014
|
Materias: | |
Acceso en línea: | https://doaj.org/article/55511374c7a244a68506538589080e25 |
Etiquetas: |
Agregar Etiqueta
Sin Etiquetas, Sea el primero en etiquetar este registro!
|
Ejemplares similares
-
Dioxin (TCDD) induces epigenetic transgenerational inheritance of adult onset disease and sperm epimutations.
por: Mohan Manikkam, et al.
Publicado: (2012) -
Developmental and lifelong dioxin exposure induces measurable changes in cardiac structure and function in adulthood
por: Matthew de Gannes, et al.
Publicado: (2021) -
Inheritable testicular metabolic memory of high-fat diet causes transgenerational sperm defects in mice
por: Luís Crisóstomo, et al.
Publicado: (2021) -
Testicular Torsion in Patients With Intellectual and Developmental Disabilities
por: Lisa B. E. Shields MD, et al.
Publicado: (2021) -
Solutions in Dioxin & Mercury Removal
por: Wirling, J.